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protein-energy malnutrition/оток
Линкът е запазен в клипборда
Страница 1 от 41 резултата
Protein-Calorie Malnutrition Requiring Revisional Surgery after One-Anastomosis-Mini-Gastric Bypass (OAGB-MGB): Case Series from the Tehran Obesity Treatment Study (TOTS).
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Adverse hemodynamic and ultrastructural changes in dog hearts subjected to protein-calorie malnutrition.
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In the absence of thiamine deficiency, the specific effects of protein-calorie malnutrition on left ventricular (L.V.) function are unknown. Mature beagle dogs of both sexes were subjected to a hypocaloric, nitrogen-poor diet which resulted in a weight loss of approximately 40% after seven weeks.
Plasma and red blood cell fatty acid composition in children with protein-calorie malnutrition.
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Plasma and red cell fatty acids were measured in 44 children with third degree malnutrition: 22 children with marasmus, 11 children with kwashiorkor, and 11 with marasmic kwashiorkor (MK). The presence of edema, dermatosis, hair changes, and hypoalbuminemia were positively correlated with the red
Serum ghrelin in infants with protein-energy malnutrition.
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BACKGROUND
Ghrelin is an appetite and weight physiologic controller. The question is whether there is a relation between ghrelin and protein-energy malnutrition (PEM). Our aim was to assess serum ghrelin in these patients and its relation to different patient variables.
METHODS
A cross-sectional
[Clinical nutritional outcome in patients recovering in a psychiatric setting from severe protein-energy malnutrition of anorexia nervosa].
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Protein energy malnutrition due to anorexia nervosa, either restrictive or bulimic, requires an integrated medical psychiatric intervention to be treated. The aim of this study was to evaluate the effectiveness of this integrated treatment in severely malnourished anorectic patients requiring to be
Acute liver injury with severe coagulopathy in marasmus caused by a somatic delusional disorder.
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Marasmus is a severe form of protein-calorie malnutrition characterized by the depletion of fat stores, muscle wasting, and the lack of edema. In developed countries, marasmus is often the result of anorexia nervosa. Abnormal transaminases with liver synthetic dysfunction have rarely been reported
Antibacterial functions of macrophages in experimental protein-calorie malnutrition. I. Description of the model, morphologic observations, and macrophage surface IgG receptors.
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An experimental protein-calorie malnutrition was produced in weanling Sprague-Dawley rats. The model resembles human malnutrition with respect to weight loss, inanition, angular stomatitis, anemia, lymphopenia, hypoproteinemia with hypoalbuminemia, and marked thymic involution. In addition, systemic
Protein-energy malnutrition as the first manifestation of cystic fibrosis in infancy.
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BACKGROUND
The protein-energy malnutrition (PEM) that is characterized by hypoproteinemia, edema, and anemia has been reported in 5-13% of infants with cystic fibrosis (CF). Due to the surprising higher incidence of PEM as the first presenting manifestation of CF in Macedonia, the aim of the present
Using of WHO guidelines for the management of severe malnutrition to cases of marasmus and kwashiorkor in a Colombia children's hospital.
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BACKGROUND
In 2007, the Hospital Infantil Los Ángeles (HILA) in Colombia implemented a slightly-modified version of the WHO guidelines for the diagnosis and management of malnutrition during childhood.
OBJECTIVE
To evaluate the efficacy of the WHO-HILA protocol in children hospitalized with severe,
Marasmus
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To maintain the physiological requirements of the body, it is essential to take a sufficient amount of micro and macronutrients; however, the overconsumption of micronutrients and macronutrients can also be harmful. As defined by the World Health Organization (WHO), malnutrition is an ‘inadequate or
Leukocyte electrolytes and sodium transport in protein energy malnutrition.
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Intracellular electrolytes and sodium transport were measured in leukocytes obtained from malnourished children. In the presence of edema, leukocyte sodium and potassium were raised. The total flux and the glycoside-sensitive portion were increased. Loss of edema was associated with reductions in
Acquired protein energy malnutrition in glutaric acidemia.
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We report a case of acquired protein energy malnutrition with associated zinc deficiency in an 18-month-old boy with type 1 glutaric acidemia. Physical examination findings included generalized nonpitting edema, widespread desquamative plaques, and sparse hair with a reddish tinge. Laboratory
Inflammatory mediators in children with protein-energy malnutrition.
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Edema is a typical sign in kwashiorkor, which is present in a subset of patients with protein-energy-malnutrition (PEM). The pathophysiology of this edema is not well established. One of the abnormalities found in kwashiorkor is reduced concentrations of antioxidants, suggesting a compromised
Blood volume (51Cr) in severe protein-calorie malnutrition.
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The intravascular volumes were determined in 42 protein-calorie-depleted Shi children, and in 14 healthy patients. The red cell volume (RCV) was determined by a radiochromium technique; the total blood volume (TBV), the plasma volume (PV), and the total circulating albumin (TCA) were derived. The
Adrenocorticosteroids and corticosteroid binding globulins in protein calorie malnutrition.
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A study of adrenocorticosteroids and corticosteroid binding globulins was undertaken in 35 malnourished children with a weight deficit of 10 to 45%. The mean corticosteroid levels 5.9 +/- 3.0 mug% were not significantly different from control values of 6.43 +/- 3.2 mug%. Steroid levels were however
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