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5 резултата
Supplementation of Blackcurrant Anthocyanins Increased Cyclic Glycine-Proline in the Cerebrospinal Fluid of Parkinson Patients: Potential Treatment to Improve Insulin-Like Growth Factor-1 Function.
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BACKGROUND
Insulin-like growth factor-1 (IGF-1) function is impaired in Parkinson disease. Cyclic glycine-proline (cGP), a metabolite of IGF-1, is neuroprotective through improving IGF-1 function. Parkinson disease patients score lower on Hospital-associated Anxiety and Depression Scale after
The complete nucleotide sequence of RNA2 of blackcurrant reversion nepovirus.
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The complete nucleotide sequence of blackcurrant reversion nepovirus (BRV) RNA2 was determined from cDNA clones. RNA2 was 6400 nucleotides (nt) in length excluding the 3' poly(A)-tail. It contained a single open reading frame of 4878 nts encoding a polypeptide of 1626 amino acids with a calculated
Physiological Responses to the Foliar Application of Synthetic Resistance Elicitors in Cape Gooseberry Seedlings Infected with Fusarium oxysporum f. sp. physali.
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Vascular wilt caused by Fusarium oxysporum is the most limiting disease that affects cape gooseberry (Physalis peruviana L.) crops in Colombia. The use of synthetic elicitors for vascular wilt management is still scarce in Andean fruit species. The objective of the present study was to
Physiological Response of Cape Gooseberry Seedlings to Three Biological Control Agents Under Fusarium oxysporum f. sp. physali Infection.
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Cape gooseberry (Physalis peruviana) fruit has gained recognition owing to its nutritional value and versatility to be consumed processed or as a fresh product. These characteristics have made it an important product in both national and international markets. One of the main limitations for
Dissection of the Drosophila paired protein: functional requirements for conserved motifs.
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The Drosophila paired gene encodes three conserved motifs: a homeodomain, paired domain and PRD (his/pro) repeat. To investigate the functional importance of the PRD repeat and paired domain, we tested deletion mutants using an ectopic expression assay in embryos. Our results suggest that the PRD
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