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antianginal/infarkt
Veza se sprema u međuspremnik
Page 1 od 525 rezultati
Reduction of size of myocardial infarction with nicorandil, a new antianginal drug, after coronary artery occlusion in dogs.
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The effects of nicorandil, a new antianginal drug, on size of myocardial infarction were studied in anesthetized, open-chest dogs after left anterior descending coronary artery occlusion. To quantify the extent of the hypoperfused zone, 99mTc-albumin microspheres were injected into the left atrium 1
Effect of antianginal drugs in stable angina on predicted mortality risk after surviving a myocardial infarction: a preliminary study (METRO).
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BACKGROUND
Although antianginal drugs are used over several months and through to years in stable angina, there is scant evidence regarding their influence on outcomes. The METRO (ManagEment of angina: a reTRospective cOhort) study sought to assess the independent effect of using these drugs on
[Antianginal activity of cardil and cordafen in patients with non-Q wave myocardial infarction].
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The antianginal activity of Cardil, 240-360 mg/day, and Cordafen, 30-60 mg/day, was evaluated in 74 patients with non-Q wave acute myocardial infarction. Repeated 24-hour ECG monitoring was used as an objective tool for the evaluation of their therapeutical efficiency in two randomized groups.
ECG gated thallium 201 myocardial images: value in detecting multivessel disease in patients on anti anginal therapy 1-3 months after myocardial infarction.
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ECG gated 201Tl scintigraphy was compared to non gated images for detecting 2-3 vessel disease 1-3 months after a first uncomplicated myocardial infarction. In all, 111 patients on anti anginal treatment underwent coronary arteriography and stress thallium imaging; 39 showed single vessel disease
[Causes of Angina Pectoris in Survivors of Myocardial Infarction and the Role of Optimization of Antianginal Therapy. Data of the LINKOR Study E..]
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to analyze causes of attacks of angina pectoris and assess impact of optimization of antianginal therapy on frequency of attacks provoked by various factors.
We analyzed diaries of 1226 survivors of myocardial infarction (MI) which occurred within 12months before inclusion in the LINKOR program.
Mildronate, a novel fatty acid oxidation inhibitor and antianginal agent, reduces myocardial infarct size without affecting hemodynamics.
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Mildronate is a fatty acid oxidation inhibitor approved as an antianginal drug in parts of Europe. We carried out the first study to determine whether a 10-day course of mildronate could reduce myocardial infarct size (IS) during acute myocardial ischemia. Sprague Dawley rats received 200 mg/kg/d of
Modification of the circadian rhythm of onset of acute myocardial infarction by long-term antianginal treatment.
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OBJECTIVE
To elucidate the mechanism of the circadian pattern of onset of acute myocardial infarction by examining the effects of prior antianginal treatment upon it.
METHODS
Retrospective analysis of clock time of the onset of acute myocardial infarction by linear modelling to define the circadian
Anti-ischemic and antianginal effects of 60 mg isosorbide-5-mononitrate in patients treated chronically after myocardial infarction.
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25 patients with stable angina pectoris after acute myocardial infarction were given single and maintenance doses of isosorbide-5-mononitrate (IS-5-MN) in a controlled-release formulation. For evaluation of the efficacy of this treatment, a score based on exercise tests and patient's complaints
Noninvasive electrocardiographic findings and plasma norepinephrine levels in patients with post-myocardial infarction receiving anti-anginal agents.
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OBJECTIVE
The aim of this study was to investigate the effects of anti-anginal agents on plasma norepinephrine (NE) levels and the autonomic nerve functions evaluated by advanced noninvasive electrocardiographic (ECG) tests in post-myocardial infarction (PMI) patients.
METHODS
The subjects were 89
Pindolol postmyocardial infarction study: evaluation of the drug's antiarrhythmic and antianginal activity.
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The study was designed to assess the antiarrhythmic and antianginal properties of pindolol as well as the tolerance to the drug by patients who had experienced a myocardial infarction 5 to 6 months previously. Exercise tests were performed in 521 postinfarct patients before and after a single oral 5
Effect of ranolazine, an antianginal agent with novel electrophysiological properties, on the incidence of arrhythmias in patients with non ST-segment elevation acute coronary syndrome: results from the Metabolic Efficiency With Ranolazine for Less Ischemia in Non ST-Elevation Acute Coronary Syndrome Thrombolysis in Myocardial Infarction 36 (MERLIN-TIMI 36) randomized controlled trial.
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BACKGROUND
Ranolazine, a piperazine derivative, reduces ischemia via inhibition of the late phase of the inward sodium current (late I(Na)) during cardiac repolarization, with a consequent reduction in intracellular sodium and calcium overload. Increased intracellular calcium leads to both
The antianginal agent, ranolazine, reduces myocardial infarct size but does not alter anatomic no-reflow or regional myocardial blood flow in ischemia/reperfusion in the rabbit.
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It has been suggested that ranolazine protects the ischemic/reperfused heart by reducing diastolic wall pressure during ischemia. However, there is limited information regarding the effect of ranolazine on the anatomic zone of no-flow in a model of acute myocardial occlusion/reperfusion. Before
Metoprolol in acute myocardial infarction. Narcotic analgesics and other antianginal drugs. The MIAMI Trial Research Group.
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The effect of metoprolol on chest pain has been assessed in terms of the duration and the use of narcotic analgesics, nitrates and calcium-channel blockers. Fewer metoprolol-treated patients in the MIAMI trial were given narcotic analgesics (49% of the placebo patients vs 44% of the metoprolol
Antianginal and antiarrhythmic effects of pindolol in post-infarct patients.
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Are antianginal drugs effective in secondary prevention after myocardial infarction?
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