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calcinosis/nikotin
Veza se sprema u međuspremnik
7 rezultati
Taurine protection against experimental arterial calcinosis in mice.
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Combined administration of vitamin D3 and nicotine produced severe myocardial and aortic calcinosis in mice within 4 days. Treatment of these mice with taurine increased the survival rate of the mice and reduced the elevation of calcium content in both aorta and myocardium as did diltiazem
Protective effects of benidipine hydrochloride (KW-3049), a calcium antagonist, against experimental arterial calcinosis and endothelial dysfunction in rats.
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Protective effects of benidipine hydrochloride (KW-3049) against arterial calcinosis and its possible mechanisms of action have been investigated. Arterial calcinosis was induced in rats by combined administration of vitamin D2 (1050000 IU/kg, s.c.) and nicotine (12.5 mg/kg, p.o., b.i.d.) for 6
Experimental basis of the long-term therapy of arterial hypertension with calcium antagonists.
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In vascular smooth muscle (as in myocardial fibers) a transmembrane supply of Ca++ ions is required for active tension development. In consequence, Ca++ antagonists, which restrict transmembrane Ca++ delivery, possess a wide scope of action against practically all types of vasoconstrictor or spastic
Haemodynamic effects of the calcium facilitator Bay K-8644 in rats following vascular calcium overload.
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1. The haemodynamic effects of the Ca2+ facilitator Bay K-8644 (Bay) were studied in a model of calcinosis induced by acute treatment with vitamin D3 and nicotine administration over 4 days with 13 days of recovery. 2. Calcium content of the left ventricular myocardium increased 8-9 fold, while
Calcium--a neglected key factor in arteriosclerosis. The pathogenic role of arterial calcium overload and its prevention by calcium antagonists.
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Using specific calcium antagonists as experimental tools, both the physiological messenger and current carrying function of calcium ions as well as their pathogenetic potencies could be elucidated. Notably, excess intracellular calcium signalling and intra- and extracellular calcium overload turned
Phytate (Myo-inositol hexakisphosphate) inhibits cardiovascular calcifications in rats.
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Calcification is an undesirable disorder, which frequently occurs in the heart vessels. In general, the formation of calcific vascular lesions involves complex physicochemical and molecular events. Calcification (hydroxyapatite) is initiated by injury and is progressed by promoter factors and/or the
Identification of Women at High Risk of Breast Cancer Who Need Supplemental Screening
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Background Mammography screening reduces breast cancer mortality, but a proportion of breast cancers are missed and are detected at later stages or develop during between-screening intervals. Purpose To develop a risk model based on negative mammograms that identifies women likely to be diagnosed
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