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diphenylhydantoin/hepatitis
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15 rezultati
Diphenylhydantoin-induced hepatitis: a case report.
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This case report is of a 25-year old man with severe diphenylhydantoin-induced hepatitis, fever, rash and generalized lymphadenopathy. Hepatic histology and lymphocyte transformation tests strongly supported the diagnosis. He made an uneventful recovery with supportive therapy.
Diphenylhydantoin sensitivity. a syndrome resembling infectious mononucleosis with a morbilliform rash and cholestatic hepatitis.
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A ten-year-old boy presented with an illness resembling infectious mononucleosis associated with an unusual skin rash and marked cholestatic hepatitis. This combination represents a rare reaction to diphenylhydantoin. Few reported cases have been published. The illness commences from three to seven
Diphenylhydantoin-induced hepatitis.
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Diphenylhydantoin sodium hepatitis.
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[Hepatitis caused by diphenylhydantoin].
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Anicteric hepatitis induced by diphenylhydantoin.
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[Chronic active hepatitis caused by diphenylhydantoin].
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Diphenylhydantoin sensitivity; report of fatal case with hepatitis and exfoliative dermatitis.
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[Toxic cholestatic hepatitis due to phenytoin].
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We report the case of a 51-years-old female who was prophylactically treated with diphenylhydantoin after surgery of an intracranial aneurysm. Twenty-four days after beginning the treatment, a general syndrome appeared, in addition to a diffuse cutaneous exanthema. Four days later, a cholestatic
Mononucleosis and hepatic failure associated with diphenylhydantoin treatment in an infant.
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Diphenylhydantoin-induced hepatitis and mononucleosis are uncommon in children. The occurrence of these two diseases in the same individual, with progression to hepatic failure is rare and has not been reported in infants. This report represents a 6-month-old male infant who developed an infectious
Hepatic injury associated with diphenylhydantoin therapy. A clinicopathologic study of 20 cases.
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Although the toxicity of diphenylhydantoin in a number of target organs is discussed in numberous reports, hepatotoxic reactions are quite rare. The hepatic lesion has mostly been described as necrosis or hepatitis. Tissue sections from 20 patients who had validated diphenylhydantoin hepatotoxicity
Lupus activation with cerebritis following pegylated interferon in a hemodialysis patient.
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BACKGROUND
A 20-year-old female patient with chronic hepatitis C virus infection and end-stage renal disease due to systemic lupus erythematosus was admitted to hospital with fever, pain in the abdomen, seizures and an alteration of consciousness. The patient was on maintenance dialysis and was
The occurrence of intramitochondrial Ca2+ granules in valproate-induced liver injury.
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Two epileptic patients treated with anticonvulsants of valproic acid, phenobarbital, nitrazepam, and diphenylhydantoin developed anorexia, convulsions, and unconsciousness. The liver biopsy samples showed degenerated hepatocytes containing enlarged mitochondria with a distorted matrix. The matrix
[Evaluation of changes in gamma-glutamyltransferase in chronic treatment with antiepileptic agents].
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To know the intensity of liver enzyme induction during a treatment with anticonvulsant, the authors have measured gamma GT before and at the 7th, 30th, 60th days after a treatment by one of the 4 major anticonvulsant as phenobarbital, diphenylhydantoin, carbamazepine and sodium valproate. All
Oxcarbazepine (GP 47 680) in the treatment of intractable seizures.
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A single blind, within-patient clinical study was carried out on sixteen profoundly mentally retarded in-patients. Five of them had primary generalized, four had partial, six secondary generalized and one had mixed epileptic seizures. With the exception of two patients, all had for months or years
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