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Subarachnoid hemorrhage presenting an "abnormal movement"--case report.
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A 61-year-old male presented with subarachnoid hemorrhage manifesting an abnormal movement as the initial symptom. The movement was rhythmic with phases: tongue protrusion with eyes wide open, and tongue retraction with eyes closed, lasting for about 10 minutes. Neuroradiological methods identified
Posttraumatic hemiballism with focal discrete hemorrhage in contralateral subthalamic nucleus.
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Although head trauma has occasionally been described as a cause of hemiballism, relevant traumatic lesion involving subthalamic nucleus (STN) has rarely been reported. We report a 49-year-old man with focal and discrete traumatic STN hemorrhage, which presented as transient contralateral
Astasia, asymmetrical asterixis and pretectal syndrome in thalamo-mesencephalic hemorrhage.
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We report a patient who presented with sudden onset instability and diplopia. On neurological examination he had asymmetrical asterixis, predominantly in the left hand, and ocular findings consistent with a pretectal syndrome. He was also unable to stand or even sit up unassisted, with a tendency to
Unilateral asterixis due to thalamic hemorrhage.
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Unilateral asterixis involving the face, hand, and foot was caused by a contralateral hypertensive thalamic hemorrhage, confirmed by computerized tomography. In addition to the asterixis, loss of upward gaze and pupillary abnormalities suggested secondary midbrain compression. These findings raise
Focal asterixis caused by a small putaminal hemorrhage.
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Unilateral asterixis and mild pure motor hemiparesis contralateral to a small putaminal hemorrhage were observed in a 74-year-old hypertensive man. The motor deficit proved to be rapidly reversible, but the focal asterixis remained for some time as the only sign of the intracranial hemorrhage. This
[Three cases of involuntary movements following pontine hemorrhage].
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We reported three cases with involuntary movements following pontine hemorrhage. All cases had various symptoms indicating brain-stem lesions, but the consciousness and motor functions were not severely disturbed. CT scans showed a small hematoma localized in unilateral pontine tegmentum in all
[Cerebellar hemorrhage and unilateral asterixis].
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A hypertensive woman presented with asterixis ipsilateral to a cerebellar hemorrhage. Asterixis has not previously been described in association with a cerebellar lesion. The presence of asterixis in this patient may have been due to a reduction of afferent information through the loss of the
Non-ketotic hyperglycemic chorea-hemiballismus mimicking basal ganglia hemorrhage.
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Radiographic findings of hyperglycemic non-ketotic chorea-hemiballismus and basal ganglia hemorrhage can be highly similar. A 58-year-old female presented with a 1-week history of choreiform and ballistic movements of the left arm. Based on CT imaging, the patient was diagnosed with a basal ganglia
[Improvement in hemiballism after transient hypoxia in a case of subthalamic hemorrhage].
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A 73-year-old man was admitted complaining of violent involuntary movement in the left upper and lower extremities. He had a ten-year history of hypertension and had had a left thalamic hemorrhage 6 years before admission. On neurological examination Horner's sign in the right eye, typical
[Hemichorea-hemiballism associated to basal ganglia hemorrhage in uncontrolled diabetes mellitus: report of two cases].
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Two cases of acute onset hemichorea-hemiballism in female patients with complicated diabetes mellitus are described. Computerized tomography showed diffuse basal ganglia hemorrhage contralateral to the abnormal movements. Occurrence of such a presentation in a teenager with insulin-dependent
[Hemiballism as a result of a focal hemorrhagic lesion of the subthalamic nucleus documented by CT].
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The authors relate a case of hemiballism with favourable evolution, suddenly arisen in an hypertensive subject, in whom the CT (Computer Tomography) pointed out a small hemorrhage at the level of the subthalamic nucleus (body of Luys), controlateral to hypercinesias. The authors underline the
MRI in hemiballismus due to subthalamic nucleus hemorrhage: an unusual complication of liver transplantation.
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A 60-year-old man developed hemiballismus due to an intracranial hemorrhage involving the subthalamic nucleus 8 weeks after orthotopic liver transplantation. The hemorrhage was thought to be due to alterations in cerebral blood flow following a period of hypotensive shock due to sepsis, in the
Development of involuntary movements after ventriculoperitoneal shunting for normal pressure hydrocephalus in a patient with chronic-phase thalamic haemorrhage.
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BACKGROUND
Delayed-onset involuntary movements have been described after thalamic stroke.
METHODS
We treated a patient with involuntary movements that increased after ventriculoperitoneal shunting (VPS) for normal pressure hydrocephalus (NPH) following thalamic haemorrage. One and one-half years
[Recurrent unipolar depression with 48-hour cycle and the state--dependent abnormal involuntary movements following right thalamic hemorrhage].
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A 70-year-old woman was admitted because of depression and abnormal involuntary movements of her left extremities. Six months before the admission, she developed left hemiparesis caused by right thalamic hemorrhage. On neurological examination, she had mild motor and sensory hemiparesis on the left
Hemiballism with leg predominance caused by contralateral subthalamic haemorrhage.
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Hemiballism is a rare movement disorder characterised by high-amplitude movements of the limbs on one side of the body. Stroke of the contralateral basal ganglia, especially the subthalamic nucleus (STN) is the most common aetiology of acute development of hemiballism. Recently, the pathophysiology
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