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hemianopsia/atrofija
Veza se sprema u međuspremnik
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[Homonymous hemianopia and posterior cortical atrophy].
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BACKGROUND
Posterior cortical atrophy (PCA) is a clinically and radiologically defined syndrome, in which predominant symptoms focus on higher visual dysfunction with progressive course and association with cortical atrophy or hypometabolism that predominates in the posterior part of the
Quantifying the pattern of optic tract degeneration in human hemianopia.
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BACKGROUND
The existence of transsynaptic retrograde degeneration (TRD) in the human visual system has been established, however the dependence of TRD on different factors such as lesion location, size and manner of lesion acquisition has yet to be quantified.
METHODS
We obtained T1-weighted
Homonymous Hemianopsia Due to Posterior Cortical Atrophy.
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A 63-year-old woman presented to her ophthalmologist complaining of reading difficulties for two years. Ophthalmological examination revealed a homonymous hemianopsia. Brain magnetic resonance imaging (MRI) scan was interpreted as normal, but positron emission tomography (PET) showed areas of
Direct and transsynaptic retrograde degeneration and optic nerve head microvascular changes in patients with hemianopia
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Purpose: To investigate optic nerve head (ONH) microvascular changes secondary to transsynaptic retrograde degeneration (TRD), comperatively with direct retrograde degeneration and healthy controls.
Statokinetic Dissociation (Riddoch Phenomenon) in a Patient with Homonymous Hemianopsia as the First Sign of Posterior Cortical Atrophy.
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We report a 60-year-old woman with posterior cortical atrophy (PCA) who presented with left homonymous hemianopsia persisting for 5 years; the patient's condition was observed using static, but not kinetic, perimetry. This statokinetic dissociation of hemianopsia, which is often called Riddoch
Imaging reveals optic tract degeneration in hemianopia.
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OBJECTIVE
To investigate whether there is transsynaptic degeneration in the human optic tract in hemianopia. To consider how the degeneration varies with duration of hemianopia and location of insult.
METHODS
Seven patients with damage to the primary visual cortex (V1), the lateral geniculate
Retinal Ganglion Cell Atrophy in Homonymous Hemianopia due to Acquired Occipital Lesions Observed Using Cirrus High-Definition-OCT.
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Purpose. To report a reduction in macular ganglion cell layer and inner plexiform layer (GCL+IPL) thickness and circumpapillary retinal nerve fiber layer (cpRNFL) thickness using spectral-domain optical coherence tomography in patients with homonymous hemianopia due to posterior cerebral artery
Retrograde degeneration of retinal ganglion cells in homonymous hemianopsia.
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BACKGROUND
The aim of this study was to demonstrate the relationship between topographic reduction in macular ganglion cell complex (GCC) thickness as detected with spectral-domain optical coherence tomography and visual field defects caused by ischemic occipital cortical injury.
METHODS
This study
Band atrophy of the optic nerve. The histology of temporal hemianopsia.
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Cross sections of the orbital portion of the optic nerve, the decussating fibers of which underwent atrophy, have a distinctive histologic pattern. Distally, from the eyeball to the midportion of the optic nerve, this pattern of atrophy consists of a broad-based nasal and a narrow-based temporal
[Pigmentary degeneration of the retina with bitemporal hemianopia].
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[Comitiality and hemianopsia. Hemispheric atrophy with occipital predominance].
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Optic atrophy with altitudinal hemianopia in neurofibromatosis.
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[Epilespy and inferior horizontal hemianopsia accompanying atrophy of the occipital lobes. (Passive ventricular dilatation of the occipital cornua].
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Homonymous quadrantanopia and sectorial optic atrophy with infarction in the territory of the anterior choroidal artery.
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Homonymous hemianopia as the first sign of posterior cortical atrophy.
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