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hyperparathyroidism/kalij

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[The role of scintigraphy with dual tracer and potassium perchlorate (99mTcO4 & KClO4/ MIBI) in primary hyperparathyroidism].

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BACKGROUND The diagnostic value of scintigraphy using a dual tracer and high-resolution neck ecotomography in the preoperative localisation of parathyroid enlargements was evaluated in a group of consecutive patients suffering from primary hyperparathyroidism (HPT) who were uniformly studied and

Increased potassium permeability in erythrocytes from patients with hyperparathyroidism.

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Parathyroid hormone (PTH) has been shown to modify Ca2+ and Na+ transport in several epithelia. The molecular mechanisms of these effects are poorly understood. We investigated here whether PTH may modify Na+ and K+ transport across the human red blood cell membrane in vitro and ex vivo. Fourteen

Prevention of experimental hyperparathyroidism by magnesium and potassium salts.

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[Experimental hyperparathyroidism and sodium-potassium metabolism in children].

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A rare case of primary hyperparathyroidism associated with primary aldosteronism and breast cancer is reported. A 44-year-old woman was admitted to our hospital to undergo surgical removal of breast cancer. She had hypertension with low serum potassium, and slightly but significantly elevated serum
OBJECTIVE The aim of this study was to investigate the risk factors of intraoperative hyperkalemia in end-stage renal disease patients undergoing parathyroidectomy (PTx) with autotransplantation (AT). METHODS Prospective observational study. METHODS Operating room of a tertiary care medical

Association of Mutations in SLC12A1 Encoding the NKCC2 Cotransporter With Neonatal Primary Hyperparathyroidism.

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Primary hyperparathyroidism with hypercalciuria has not been described in the newborn period. Our objectives are to identify the genetic basis for neonatal primary hyperparathyroidism in a family with 2 affected children. An African American boy presenting with mild neonatal primary

Primary hyperparathyroidism and coexisting nephrogenic diabetes insipidus: rapid postoperative correction.

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Shortly after diagnosis of primary hyperparathyroidism, a patient had serum hyperosmolality, polyuria, isosthenuria, profound potassium depletion, and elevated plasma antidiuretic hormone levels, all consistent with nephrogenic diabetes insipidus. After parathyroidectomy, serum calcium and serum
BACKGROUND The extent of contribution of disturbed magnesium balance to mortality remains unclear among hemodialysis patients. METHODS This was a cohort study involving 3276 patients on maintenance hemodialysis at 86 facilities in Japan from 2008 to 2010 who had secondary hyperparathyroidism (SHPT).

Familial isolated primary hyperparathyroidism--a multiple endocrine neoplasia type 1 variant?

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OBJECTIVE Familial isolated primary hyperparathyroidism (FIHP) is defined as hereditary primary hyperparathyroidism without the association of other diseases or tumors. Linkage analyses suggest that different genotypes can lead to the same phenotype of primary hyperparathyroidism. Hereditary
The main purposes of this study were: (a) to investigate the efficacy of an imaging protocol based on the combination of 99mTcO4/MIBI scintigraphy and neck ultrasound (US) in selecting patients with primary hyperparathyroidism (HPT) for unilateral neck exploration, and (b) to help define the role of
The main purposes of this study were: (a) to investigate the efficacy of an imaging protocol based on the combination of (99m)TcO4/MIBI scintigraphy and neck ultrasound (US) in selecting patients with primary hyperparathyroidism (HPT) for unilateral neck exploration, and (b) to help define the role

Change in serum bicarbonate during acute EDTA infusions in post-transplant hyperparathyroidism.

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We evaluated the effect of endogenous parathyroid hormone (PTH) on serum bicarbonate in 12 patients with postrenal transplant hyperparathyroidism. EDTA-induced hypocalcemia produced a significant rise in serum PTH (p less than 0.001). Associated with the increase in PTH was a significant fall in
We evaluated an African American woman referred in 1986 at age 33 years because of renal potassium and calcium wasting and chronic hip pain. She presented normotensive, hypokalemic, hypocalcemic, normophosphatemic, and hypercalciuric. Marked hyperparathyroidism was evident. Urinary cyclic adenosine

X-ray microanalysis of elemental changes in human parathyroid glands in primary and secondary hyperparathyroidism.

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The elemental composition of chief cells of parathyroid glands from patients with adenomatous primary hyperparathyroidism (HPT) and uremic secondary HPT was studied by X-ray microanalysis. Glands histologically deemed normal were used as controls. The analyses were also carried out on tissue
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