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polycythemia vera/albumin
Veza se sprema u međuspremnik
10 rezultati
Can ischemia-modified albumin be a valuable indicator of tissue ischemia in polycythemia vera?
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The red cell expansion in polycythemia vera (PV) causes hyperviscosity affecting blood flow, which plays a major role in the pathogenesis of both microcirculatory disturbances and ultimately thromboses. Ischemia-modified albumin (IMA) is produced during an ischemic states and is present in blood in
Stimulus-specific defect in oxidative metabolism of polymorphonuclear granulocytes in polycythemia vera.
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We investigated polymorphonuclear granulocyte (PMN) function in polycythemia vera (PV) in relation to healthy controls. PMN oxidative metabolism, assessed by chemiluminescence (CL), was significantly lower in PV patients after stimulation with leukotriene B4 (LTB4) and f-Met-Leu Phe (fMLP) (60% of
Cell separation, cell differential and granulocyte colony frequency in polycythemia vera.
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In seven patients with polycythemia vera, the agar colony growth of bone marrow total nucleated cell suspensions and of the cell fractions obtained with an albumin discontinuous density gradient were studied. In one patient, the density distribution of colony-forming units in culture (CFUc) before
An unusual cause of hypercalcemia in polycythemia vera: parathyroid adenoma.
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In this paper we describe a patient with polycythemia vera (PV), who presented with hypercalcemia due to a parathyroid adenoma. In November 1999, the patient was admitted to our hospital with meteorism and constipation. Her physical examination revealed plethora and hepatosplenomegaly. Laboratory
Histopathological manifestations of membranoproliferative glomerulonephritis and glomerular expression of plasmalemmal vesicle-associated protein-1 in a patient with polycythemia vera.
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Only a few cases of various glomerulonephropathies have been reported in patients with polycythemia vera. We report the case of a 72-year-old female with polycythemia vera in whom renal biopsy examination showed membranoproliferative glomerulonephritis (MPGN)-like lesion and glomerular expression of
Leukocytosis in polycythemia vera and splenomegaly in essential thrombocythemia are independent risk factors for hemorrhage.
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BACKGROUND
Long-term outcomes are favorable for patients with polycythemia vera (PV) and for patients with essential thrombocythemia (ET). However, hemorrhage is a significant cause of morbidity and mortality in those patients.
METHODS
We retrospectively recruited 247 patients who had received a
Physiological changes in blood volume.
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Measurement of blood volume is an important clinical tool in establishing a diagnosis as in polycythemia vera, in assessing the true significance of a low blood count in a patient with splenomegaly, and in evaluating a bleeding patient. In theory, blood volume measurements should be of great value
Should whole-body red cell mass be measured or calculated?
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The whole-body volume of red blood cells must be known for correct diagnosis of polycythemia vera. Since the venous hematocrit may not correctly reflect the absolute amount of red blood cells, the red cell mass (RCM) is usually determined by radioisotope labeling of red blood cells according to
Red cell mass and plasma volume measurements in polycythemia: evaluation of performance and practical utility.
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BACKGROUND
Despite the absence of any systematic evidence for diagnostic utility, red cell mass (RCM) measurement has been endorsed as a major diagnostic criterion for polycythemia vera (PV) based on a set of eligibility criteria for a clinical trial formulated by an International PV Study Group in
Mobilization of granules in neutrophils from patients with myeloproliferative disorders.
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Neutrophil granule subsets and dynamics were studied in 4 patients with polycythemia vera/myelofibrosis and 2 patients with chronic myelogenous leukemia. Alkaline phosphatase, a marker for the membrane of secretory vesicles (the most readily mobilizable pool of intracellular membranes in
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