pseudorabies/edema

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Corticosteroids are unable to protect against pseudorabies virus-induced tissue damage in the developing brain.

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After intraocular injection of the virulent pseudorabies virus (PRV) strain Becker into late-stage chicken embryos, the virus spreads and replicates in the brain, where severe edema and hemorrhaging follow. By contrast, the attenuated Bartha strain does not cause severe brain pathology despite viral

Pseudorabies in a dog.

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Pseudorabies was diagnosed in a 5-year-old female crossbred dog by histologic examination and virus isolation. The clinical signs were depression, salivation, head pressing, and emesis. There were no gross pathologic findings. The microscopic findings in the brain stem consisted of mononuclear cell

Identification of neuroanatomic circuits from spinal cord to stomach in mouse: retrograde transneuronal viral tracing study.

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To determine the spinal innervation and neuronal connections is important for studying gastric carbohydrate metabolism and motor responses. Neurons involved in the efferent control of the stomach were identified following visualization of pseudorabies virus (PRV)-614 retrograde tracing. PRV-614 was

A chicken embryo eye model for the analysis of alphaherpesvirus neuronal spread and virulence.

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We describe use of developing chicken embryos as a model to study neuronal spread and virulence of pseudorabies virus (PRV). At embryonic day 12, beta-galactosidase-expressing PRV strains were injected into the vitreous humor of one eye, and virus replication and spread from the eye to the brain

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