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varicose ulcer/proline
Veza se sprema u međuspremnik
4 rezultati
Dermal fibroblasts from venous ulcers are unresponsive to the action of transforming growth factor-beta 1.
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Failure to reepithelialize is the major clinical problem in venous ulcers. It is not clear whether the problem resides with keratinocytes or with inadequate and improper formation of extracellular matrix. In this study, we characterized the biosynthetic activity and response to transforming growth
A new protocol for the treatment of the chronic venous ulcers of the lower limb.
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Venous leg ulcer is a pathological condition afflicting prevalently elderly patients, which has been found to have a major impact on individuals' health and social aspects of quality of life. Actually, the best practice treatment is recommended to include wound dressing and multilayer compression
Deregulation of keratinocyte differentiation and activation: a hallmark of venous ulcers.
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Epidermal morphology of chronic wounds differs from that of normal epidermis. Biopsies of non-healing edges obtained from patients with venous ulcers show thick and hyperproliferative epidermis with mitosis present in suprabasal layers. This epidermis is also hyper-keratotic and parakeratotic. This
Hyperglycemia regulates hypoxia-inducible factor-1alpha protein stability and function.
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Hyperglycemia and hypoxia are suggested to play essential pathophysiological roles in the complications of diabetes, which may result from a defective response of the tissues to low oxygen tension. In this study, we show that in primary dermal fibroblasts and endothelial cells, hyperglycemia
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