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endometriosis/prolina
L'enllaç es desa al porta-retalls
13 resultats
The proline form of p53 codon 72 polymorphism is associated with endometriosis.
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OBJECTIVE
To evaluate the association between endometriosis and the p53 polymorphism.
METHODS
Prospective study.
METHODS
Department of gynecology and genetics in a medical center.
METHODS
Women with and without endometriosis.
METHODS
Women were categorized as having moderate or severe endometriosis
Association of TP53 gene codon 72 polymorphism with endometriosis in Mexican women.
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The TP53 tumor suppressor gene plays an important role in cell cycle regulation; polymorphisms of this gene have been associated with endometriosis. We examined the role of TP53 codon 72 polymorphism by comparing genotypes of 235 healthy Mexican women (controls with surgically excluded
Analysis of the codon 72 polymorphism of the TP53 gene in patients with endometriosis.
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Endometriosis is a benign gynaecologic disease that is associated with a certain risk for malignant degeneration. The disease has a genetic background, but the locations of possible genomic aberrations are still poorly clarified. In this context, the proline form of TP53 codon 72 polymorphism has
Association of TP53 gene codon 72 polymorphism with endometriosis risk in Isfahan.
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BACKGROUND
Endometriosis is a female health disorder that occurs when cells from the lining of the uterus grow in other areas of the body. The cause of endometriosis is unknown.
OBJECTIVE
The purpose of this study was to investigate TP53 gene codon 72 polymorphism in women with endometriosis and
Dopamine receptor D2 genotype (3438) is associated with moderate/severe endometriosis in infertile women in Brazil.
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OBJECTIVE
To compare the prevalence of dopamine receptor D2 polymorphisms in patients with peritoneal endometriosis and in healthy control subjects.
METHODS
Case-control study.
METHODS
University hospital.
METHODS
One hundred seven women aged ≥18 years who were enrolled when seeking care for
Analysis of p53 codon 72 gene polymorphism in Brazilian patients with endometriosis.
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We examined the frequency of p53 codon 72 polymorphism in 38 patients with endometriosis whose diagnosis was confirmed using videolaparoscopy. Half of the women were infertile. There were no significant differences in the genotype (P = 0.0927) or allele frequencies (P = 0.1430) for p53 Arg72Pro
Association of TP53 codon 72 polymorphism in women suffering from endometriosis from Lahore, Pakistan.
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OBJECTIVE
To investigate TP53 gene codon 72 polymorphism in women with endometriosis and compare it with healthy samples.
METHODS
This case-control study was carried out at Jinnah Hospital, Services Hospital and Sheikh Zayed Hospital, Lahore, Pakistan, from 2014 to 2016, and comprised patients with
[Effects of Bushen Wenyang Huayu Recipe on Expressions of HIF-1α, PHD2, and VHL in Endometriosis Rats with Shen Yang Deficiency Blood Stasis Syndrome].
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OBJECTIVE
To observe the effect of Bushen Wenyang Huayu Recipe (BWHR) on hypoxia inducible factor-1α (HIF-1α), proline hydroxylase2 (PHD2), von Hippel Lindau disease (VHL) suppressor gene expressions in endometriosis (EM) rats with Shen yang deficiency blood stasis syndrome (SYDBSS), and to explore
Polymorphism at codon 72 of the p53 gene is not associated with endometriosis in a Japanese population.
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OBJECTIVE
Endometriosis is inherited as a complex trait, which means that multiple susceptibility genes interact with each other and the environment to produce the phenotype. Previous studies have implicated p53, a tumor suppressor gene, as a factor in the development of the disease. In a Japanese
Metabolomic profiling and biochemical evaluation of the follicular fluid of endometriosis patients.
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Diseases are complex systems that can be studied through the integration of data derived from different disciplines to obtain a global and reliable picture of the biological phenomenon under investigation. Based on the recent observations that the metabolomics profiling of follicular fluids reflects
Expression profiling of endometrium from women with endometriosis reveals candidate genes for disease-based implantation failure and infertility.
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Endometriosis is clinically associated with pelvic pain and infertility, with implantation failure strongly suggested as an underlying cause for the observed infertility. Eutopic endometrium of women with endometriosis provides a unique experimental paradigm for investigation into molecular
Metabolomics reveals perturbations in endometrium and serum of minimal and mild endometriosis.
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Endometriosis is a common benign gynecological disease, characterized by growth and proliferation of endometrial glands and stroma outside the uterus. With studies showing metabolic changes in various biofluids of endometriosis women, we have set upon to investigate whether endometrial tissue show
Calpain7 impairs embryo implantation by downregulating β3-integrin expression via degradation of HOXA10.
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Endometriosis (ENDO) is a common gynecological disease that causes infertility in many women. Previous studies noted that the dysregulation of Homeo box A10 (HOXA10) in the endometrium of women with ENDO was involved in the failure of embryo implantation. However, the mechanism by which HOXA10
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