epilepsia partialis continua/infart

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Palatal tremor as a manifestation of epilepsia partialis continua caused by acute precentral gyral infarction.

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We describe a patient with palatal tremor (PT) as a manifestation of focal seizure caused by acute cortical infarction. Brain MRI showed acute infarction in the left precentral gyrus without evidence of brainstem lesions or hypertrophy of the inferior olivary nucleus. We discuss the differences

Thyrotoxic hypercoagulable state with cerebral venous thrombosis and venous infarction masquerading as epilepsia partialis continua.

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Rubral tremor after thalamic infarction in childhood.

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The occurrence of tremor after thalamic lesions is well known. Delayed rubral tremor secondary to bilateral thalamic infarction is a rare finding and has not been reported previously in childhood. We present two children with a combined resting-postural-kinetic tremor caused by bithalamic

Focal status epilepticus and epilepsia partialis continua in adults and children.

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Focal status epilepticus and epilepsia partialis continua (FSE-EPC) are most frequently seen with chronic focal progressive encephalitis of Rasmussen and Russian spring-summer encephalitis. FSE-EPC may be the presenting feature of nonketotic hyperglycemic diabetes mellitus but is more often noted as

[Epilepsia partialis continua (Kojevnikov's syndrome)].

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BACKGROUND Lesions close to the central sulcus may give rise to focal motor seizures of long duration. This condition is called epilepsia partialis continua (Kojevnikov's syndrome). METHODS Over the last two years, the National Centre for Epilepsy in Norway has treated 12 patients with epilepsia

Focal neuronal loss, reversible subcortical focal T2 hypointensity in seizures with a nonketotic hyperglycemic hyperosmolar state.

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BACKGROUND Neuroimaging in seizures associated with nonketotic hyperglycemia (NKH) is considered normal. We report magnetic resonance imaging (MRI) abnormalities in four patients with NKH and seizures. METHODS We prospectively evaluated clinical and radiological abnormalities in four patients with

Seizures in hyperglycemic patients.

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OBJECTIVE To study the pattern of seizures in hyperglycemic patients and its correlation with serum osmolality, blood sugar and serum sodium. METHODS (Study period June 1999-June 2000) Forty patients who presented with first time seizures and who were detected to be diabetic were included in the

Distinctive FDG-PET/CT Findings in Acute Neurological Hospital Care.

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A compilation of 6 distinctive ¹⁸F-fluorodeoxyglucose positron emission tomography (PET) combined with computed tomography (CT) findings in the acute setting of neurohospital care is presented. In case 1, PET/CT allowed the final diagnosis of circumscribed ischemic infarction by

Myoclonus epilepsy in two brothers. Clinical features and neuropathology of a unique syndrome.

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We report 2 brothers with progressive ataxia, seizures, myoclonus, supranuclear ophthalmoplegia, progressive visual loss and embolic strokes. The epilepsy and myoclonus came on many years after the onset of the ataxia. In the more severely affected brother the myoclonus was often unilateral and

Opercular myoclonic-anarthric status epilepticus.

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We report 3 cases of opercular myoclonic status epilepticus (OMASE), characterized by fluctuating cortical dysarthria without true aphasia associated with epileptic myoclonus involving bilaterally the glossopharyngeal musculature. In this syndrome, the inferior rolandic area of either one or the

Border zone ("watershed area") cerebral ischemia.

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Twenty subjects showing clinical and electroencephalographic patterns of the so-called "diffuse cortical ischemic syndrome with an extraterritorial (border zone) predilection," as described by Gastaut and Naquet (1965) and Gastaut et al. (1971) were studied. This syndrome occurs in elderly patients

Seizures in an immunocompromised adolescent: a case report.

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BACKGROUND Tuberculosis is a progressive and disabling infection predominantly seen in low-income and middle-income countries. Immunocompromised patients are at a higher risk of contracting tuberculosis than the healthy population. The presentation may also be atypical, leading to delay in

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