hemifacial spasm/inflamació

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A correlative analysis between inflammatory cytokines and trigeminal neuralgia or hemifacial spasm.

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It is necessary to understand the mechanism of trigeminal neuralgia (TN) and hemifacial spasm (HFS) in order to seek for an effective noninvasive remedy. As previous studies implied that inflammatory cytokines induced by demyelination following the nerve injury may be the initiated

Preliminary Study on the Relationship Between Inflammation and Hemifacial Spasm.

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Complete pathogenesis of hemifacial spasm (HFS) is unknown. In some patients with HFS, thickened, opaque, and sticky arachnoid membranes have been found during microvascular decompression procedures. This phenomenon indicates a possible relationship between inflammation and HFS. The

Multiple Sclerosis Presenting with Facial Twitching (Myokymia and Hemifacial Spasms).

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Multiple sclerosis (MS) is a chronic inflammatory demyelinating disease of the central nervous system. The etiology is insufficiently understood. Autoimmune, genetic, viral, and environmental factors have been hypothesized. MS is twice as common in women as in men between the ages of 20 and 50

Hemifacial spasm: case report.

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A 53 year old lady with diabetes mellitus presented with right hemi-facial spasm (HFS). Brain MRI Scan revealed extensive pan-sinusitis and mild bilateral mastoiditis. She responded well to intravenous ceftriaxone therapy and the hemifacial spasm resolved. The symptoms of hemifacial spasm and pain

Placement of Teflon Sponges in Microvascular Decompression Procedure for Treatment of Hemifacial Spasm.

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Background Hemifacial spasm (HFS) is generally treated by microvascular decompression (MVD). Inadequate separation of vessel and nerve or adhesive inflammation surrounding the nerve root may cause recurrence. Objective To explore a method to reduce the incidence of adhesions and to ensure sufficient

Changes in neurotrophic and inflammatory factors in the cerebrospinal fluid of patients with postherpetic neuralgia.

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Inflammatory and neurotrophic factors are involved in postherpetic neuralgia (PHN), but the association of these factors in the cerebrospinal fluid (CSF) with the level of pain is poorly known. The present study aimed to examine the changes in neurotrophic and inflammatory factors in the CSF of

Long-term efficacy of local doxorubicin chemomyectomy in patients with blepharospasm and hemifacial spasm.

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OBJECTIVE This study examines the long-term follow-up of all patients treated with doxorubicin injections in the eyelids. METHODS Nonrandomized clinical trial. METHODS Eighteen patients with blepharospasm (12 female; 6 male) and nine patients with hemifacial spasm (4 female; 5 male). METHODS Eyelids

[Delayed facial palsy after microvascular decompression for hemifacial spasm due to reactivation of varicella-zoster virus].

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We report a patient suffering from delayed facial palsy after microvascular decompression (MVD) for hemifacial spasm, in whom the pathogenesis was proved. A 56-year-old man with a left hemifacial spasm was admitted to our hospital. Preoperative MR imaging showed that the left anterior inferior

Hemifacial spasm in tuberculous meningitis.

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Hemifacial spasm developed in a 47-year-old man with tuberculous meningitis. The spasms ceased completely following vigorous antituberculous treatment. A selective compression of the facial nerve root along its exit at the brainstem by a localised inflammatory process is the most tenable explanation

[Vascular, inflammatory and tumorous lesions of the temporal bone and cerebellopontile angle: applications of magnetic resonance tomography].

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Advances in high resolution magnetic resonance imaging (MRI) allow the evaluation of the cerebellopontine angle and temporal bone. The purpose of our study was to demonstrate pathological and anatomical aberrations of cerebral arteries in relation to clinical symptoms. The study was carried out on a

Local injections of corticotropin releasing factor reduce doxorubicin-induced acute inflammation in the eyelid.

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OBJECTIVE Doxorubicin chemomyectomy is an effective alternative treatment option for patients with blepharospasm and hemifacial spasm. One side effect of the use of doxorubicin in localized injections is the development of acute inflammation and skin injury at the injection site. Corticotropin

Revision Microvascular Decompression for Trigeminal Neuralgia and Hemifacial Spasm: Factors Associated with Surgical Failure.

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Objective To investigate risk factors for symptom recurrence in patients requiring a revision microvascular decompression (MVD) for trigeminal neuralgia (TN) or hemifacial spasm (HFS). Design Retrospective review of a prospectively maintained database. Participants Seventeen

Bilateral reversible basal ganglia changes associated with dystonia and hemifacial spasms in central nervous system lupus.

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We report a 40-year-old woman with systemic lupus erythematosus (SLE) and associated inflammatory polyarthritis who presented with acute facial dystonic spasms. Her speech was also affected. An MRI brain showed bilateral symmetrical basal ganglia signal change on T2. This movement disorder was due

Microvascular decompression for the patient with painful tic convulsif after Bell palsy.

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Painful tic convulsif is referred to as the concurrent trigeminal neuralgia and hemifacial spasm. However, painful tic convulsif after ipsilateral Bell palsy has never been reported before. We report a case of a 77-year-old woman with coexistent trigeminal neuralgia and hemifacial spasm who had

[Cholesterol granuloma].

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OBJECTIVE We report 2 cases (52 and 29-year-old men) of CG in the petrous apex we treated in 2004-2006. We also review the literature and discus the mechanism of development of CG and the treatment of this lesion. METHODS Cholesterol granuloma (CG) is a chronic inflammatory process with associated

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