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myokymia/inflamació
L'enllaç es desa al porta-retalls
14 resultats
Teaching Video NeuroImages: Widespread clinical myokymia in chronic inflammatory demyelinating polyradiculoneuropathy.
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Facial Myokymia Due to a Pontine Inflammatory Demyelinating Disorder Associated with Mixed Connective Tissue Disorder [MCTD]
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Multiple Sclerosis Presenting with Facial Twitching (Myokymia and Hemifacial Spasms).
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Multiple sclerosis (MS) is a chronic inflammatory demyelinating disease of the central nervous system. The etiology is insufficiently understood. Autoimmune, genetic, viral, and environmental factors have been hypothesized. MS is twice as common in women as in men between the ages of 20 and 50
Superior oblique myokymia: a prism lens approach to control.
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A clinical evaluation of a 35-year-old man with episodic vertical diplopia and oscillopsia is discussed. A diagnosis of superior oblique myokymia, a relatively rare and frequently undiagnosed neuromuscular syndrome is made. Considerable medical research suggests that this is a motor neuron
Rituximab in the treatment of three coexistent neurological autoimmune diseases: chronic inflammatory demyelinating polyradiculoneuropathy, Morvan syndrome and myasthenia gravis.
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A 76-year-old man with a pre-existing diagnosis of myasthenia gravis was admitted to an intensive care unit with pneumonia and type II respiratory failure. In addition, muscle weakness, widespread myokymia, neuropsychiatric disturbance and autonomic disturbance were present. Antivoltage gated
[Multifocal polyneuropathy with persistent conduction blockage. A new subset of chronic inflammatory polyneuropathies].
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Recently a subset of chronic demyelinating inflammatory polyneuropathies with asymmetrical involvement limited to upper limbs, at least at the onset, resembling a multifocal mononeuropathy has been described. Electrodiagnostic testing disclosed multifocal CB outside the common entrapment sites has
[Morvan's fibrillary chorea].
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Two cases of Morvan's chorea are reported. One of the patients presented the characteristic of having had two attacks, the first after organic mercury preparations, and the second after gold salts for inflammatory rheumatism. The second case had facial fibrillations only, and this was followed by a
Isaacs' syndrome associated with chronic inflammatory demyelinating polyneuropathy.
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We report the first case of Isaacs' syndrome in which an inflammatory demyelinating neuropathy was documented histologically. For 9 months, the patient developed slowly progressive weakness, muscle spasms and stiffness, fasciculations, and myokymia in the arms, which were unmodified by sleep. Nerve
Limb myokymia.
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Thirty-eight patients with myokymic discharges localized to limb muscles on needle electromyography had various neurologic lesions, both acute and chronic. Of the 38 patients, 27 had had previous radiation therapy and the clinical diagnosis of radiation-induced plexopathy, myelopathy, or both. For
The stiff-man syndrome and related disorders.
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The stiff-man syndrome (SMS) is characterised by rigidity and spasm of predominantly axial and proximal limb muscles. The cause of the condition is unknown but the finding of antibodies to glutamic acid decarboxylase (GAD) in approximately 60% of patients has suggested an autoimmune basis.
Isaac's syndrome associated with CIDP and pregnancy.
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Neuromyotonia with all its synonyms is a disorder of peripheral nerve hyperexcitability characterized by regular or irregular myokymia, muscle cramps and stiffness, delayed muscle relaxation after contraction, and hyperhidrosis associated with well-described spontaneous electromyographic features.
Morvan's syndrome and myasthenia gravis related to familial Mediterranean fever gene mutations.
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BACKGROUND
We present the first case of Morvan's syndrome (MoS) and myasthenia gravis (MG) related to familial Mediterranean fever (FMF) gene mutations.
METHODS
A 40-year-old woman with a 1-year history of bilateral ptosis and limb muscle weakness presented to our hospital. She also had memory
A 6-Year-Old With Leg Cramps.
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A 6-year-old girl presented with a history of leg pain and cramping that progressively worsened over a 2- to 3-week period of time. Her examination was notable for normal vital signs, limited range of motion of her left hip, and a limp. Inflammatory markers were slightly elevated, but the serum
Calcium effect on generation and amplification of myokymic discharges.
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Maneuvers designed to manipulate ionized calcium (Ca++) were carried out in two patients with inflammatory polyradiculoneuropathy and myokymia. Increased clinical myokymia and myokymic burst amplification occurred when ionized Ca++ was lowered by plasma exchange or hyperventilation. Increasing
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