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nephrosclerosis/potassium
L'enllaç es desa al porta-retalls
Pàgina 1 des de 29 resultats
Serum potassium predicts time to blood pressure response among African Americans with hypertensive nephrosclerosis.
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It is not known whether serum potassium levels affect blood pressure response to antihypertensive medication. The African American Study of Kidney Disease and Hypertension (AASK) Genomics Study (N=828) is a subset of the AASK trial that randomized 1094 African American men and women with
Dexamethasone suppressible hyperaldosteronism in a child with nephrosclerosis.
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A 9 year old Mexican boy presented with severe hypertension, hypokalaemia and features suggesting acute glomerulonephritis. Nephrosclerosis was present on renal biopsy. Aldosterone levels were unresponsive to variations in dietary salt intake and plasma renin activity was suppressed. Following oral
Idiopathic Recurrent Calcium Urolithiasis (IRCU): pathophysiology evaluated in light of oxidative metabolism, without and with variation of several biomarkers in fasting urine and plasma--a comparison of stone-free and -bearing male patients, emphasizing mineral, acid-base, blood pressure and protein status.
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BACKGROUND
IRCU is traditionally considered as life?style disease (associations with, among others, overweight, obesity, hypertension, type-2 diabetes), arising from excess, in 24 h urine, of calcium (Ca) salts (calcium oxalate (CaOx), calcium phosphate (CaPi)), supersaturation of, and
Liver-derived IGF-I regulates kidney size, sodium reabsorption, and renal IGF-II expression.
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The GH/-IGF-I axis is important for kidney size and function and may also be involved in the development of renal failure. In this study, the role of liver-derived endocrine IGF-I for kidney size and function was investigated in mice with adult liver-specific IGF-I inactivation (LI-IGF-I(-/-) mice).
Sodium restriction and blood pressure in hypertensive type II diabetics: randomised blind controlled and crossover studies of moderate sodium restriction and sodium supplementation.
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OBJECTIVE
To determine the effect of moderate dietary sodium restriction on the hypertension of non-insulin-dependent (type II) diabetes.
METHODS
Randomised parallel controlled study of moderate sodium restriction for three months compared with usual diabetic diet, followed by randomised double
Does high salt intake cause hyperfiltration in patients with essential hypertension?
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In animal models of salt-dependent hypertension, hyperfiltration is associated with a faster decline in renal function and there is evidence that in hypertensive man, increased creatinine clearance is a marker of early hypertensive nephropathy. We have studied the influence of salt intake on the
Endothelin-aldosterone interaction and proteinuria in low-renin hypertension.
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OBJECTIVE
To investigate whether endothelin and aldosterone participate in the increased prevalence and severity of nephrosclerosis in human low-renin hypertension, analogous to observations in experimental hypertension.
METHODS
Comparison of endothelin, aldosterone and their relationships with
Study of the association of -667 aquaporin-2 (AQP-2) A/G promoter polymorphism with the incidence and clinical course of chronic kidney disease in Korea.
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BACKGROUND
Impaired urinary concentration is uniformly present with advanced disease in chronic renal failure. Aquaporin-2 (AQP-2) is known to be expressed in the renal collecting duct cells and participates in urinary concentration in response to vasopressin. Recently, the study of AQP expression
Long-term results of surgical and conservative treatment of patients with primary aldosteronism.
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The long-term results of surgical and specific drug therapy were compared in a group of 57 patients with primary aldosteronism (PA) (46 with aldosterone-producing adenoma (APA), 11 with idiopathic hyperaldosteronism (IHA) and bilateral adrenal hyperplasia). Unilateral adrenalectomy completely
Effect of add-on direct renin inhibitor aliskiren in patients with non-diabetes related chronic kidney disease.
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BACKGROUND
The renin-angiotensin-aldosterone system (RAAS) plays an important role in the progression of chronic kidney disease (CKD). Although dual RAAS inhibition results in worse renal outcomes than monotherapy in high risk type 2 diabetes patients, the effect of dual RAAS inhibition in patients
Renal involvement in leprosy: evaluation of patients in Turkey.
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BACKGROUND
Renal involvement in leprosy has previously been described in the literature and can include amyloidosis, glomerulonephritis, nephrosclerosis, tubulointerstitial nephritis, and granulomas.
OBJECTIVE
To evaluate renal involvement in Turkish patients with leprosy.
METHODS
In total, 32
Differences in blacks and whites with essential hypertension: biochemistry and endocrine. State of the art lecture.
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Overall, there is agreement that the origins of hypertension have a genetic basis. The genetic factors interact with environmental factors that influence expression and intensity of the disorder. As summarized in Table 1, there is evidence from the literature to identify pathways for the development
Effects of nicardipine on blood pressure and renal function in elderly hypertensive patients with renal dysfunction.
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The effects of monotherapy with nicardipine, 20 mg three times a day, have been investigated in a 1-year study of 26 elderly (greater than 60 years) patients with hypertension with various types of renal dysfunction and seven without renal dysfunction. Parameters measured included blood pressure,
Mineralocorticoid receptor activation and blockade: an emerging paradigm in chronic kidney disease.
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Slowing the progression of chronic kidney diseases (CKDs) requires new and effective treatment approaches. Aldosterone classically acts on the distal nephron: it facilitates sodium reabsorption, potassium secretion, and participates in blood pressure control. Recently, new targets of aldosterone
Interactions of PPAR α and GLUT4 in DOCA/salt-induced renal injury in mice.
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Diminished insulin sensitivity is a characteristic feature of various pathological conditions such as hypertension and activation of peroxisome proliferator activated receptor α (PPARα) has been shown to enhance insulin resistance and reduce capacity for glucose uptake in muscles. The present study
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