pseudobulbar palsy/astènia

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Thoracic dural arteriovenous fistula presenting with isolated pseudobulbar palsy mimicking brainstem lesion: A case report.

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Spinal dural arteriovenous fistulas (DAVF) are usually associated with neurological dysfunction adjacent to the shunt point; however, the symptoms are uncommon far from the site of fistula. To our knowledge, this is the first report of a patient with rapidly progressive isolated

Hashimoto's encephalopathy presenting as pseudobulbar palsy.

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BACKGROUND Hashimoto's encephalopathy (HE) is an autoimmune condition with varied neurological and psychiatric features. HE is very unusual as a cause of pseudobulbar palsy (PSP). METHODS A 14-year-old male was admitted with right-sided weakness, dysphagia, speech disorder, and aggressiveness. Brain

Pseudobulbar Palsy

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Magnus reported the first case of pseudobulbar palsy in 1837 in a patient having multiple infarcts. Lepine, in 1877 introduced the term pseudobulbar palsy for differentiation purposes. Pseudobulbar palsy is due to an upper motor lesion caused by bilateral disturbance of the corticobulbar tracts. The

Acute Pseudobulbar Palsy After Bilateral Paramedian Thalamic Infarction: A Case Report.

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Bilateral paramedian thalamic infarction is a rare subtype of stroke caused by occlusion of the artery of Percheron, an uncommon variant originating from one of the posterior cerebral arteries. This type of stroke has several major clinical presentations: altered mental status, behavioral amnestic

[Dementia in progressive supranuclear paralysis patients].

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BACKGROUND Progressive supranuclear paralysis is difficult to detect in its initial stages. Its symptoms are not specific and often the patients are mistaken as Parkinson s disease patients, or even Alzheimer s ones. METHODS Initial features in progressive supranuclear paralysis are typically a gait

[A case of variant Gerstmann-Sträussler-Scheinker disease with the mutation of codon P105L].

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Here we present a case of variant GSS disease with mutations in codons 1055 and 129 in a prion protein. The patient was a 54-year-old male, who developed weakness in the lower limbs and spastic, wide-based gait at the age of 46 years. Subsequently he developed dementia and spastic quadriplegia at

An adult-onset case of sporadic motor neuron disease with basophilic inclusions.

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A 36-year-old man developed motor neuron signs consisting of weakness and atrophy of the right upper limb, which progressed to involve the other limbs along with development of upper motor neuron signs including pseudobulbar palsy. He died 8.5 years after onset. Bilateral precentral gyri and

Rapidly progressive atypical parkinsonism associated with frontotemporal lobar degeneration and motor neuron disease.

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OBJECTIVE To report the rare but distinct clinical and neuropathological phenotype of non-familial, rapidly progressive parkinsonism and dementia associated with frontotemporal lobar degeneration with motor neuron disease (FTLD-MND). METHODS Subjects included two 70-year-old women presenting with

Familial childhood primary lateral sclerosis with associated gaze paresis.

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Three children from consanguineous parents began losing the ability to walk in late infancy. Despite chronically progressive weakness leading to wheelchair dependence by adolescence and later loss of motor speech production, intellect remained preserved. Examination revealed upper motor neuron

Facial paralysis in children: differential diagnosis.

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The differential diagnosis in 170 patients between birth and 18 years of age is reviewed. There are a number of obvious physical findings and historical features that allow one to make a diagnosis rather quickly. Pain, vesicles, a red pinna, vertigo, and sensorineural hearing loss suggest herpes

[Transitory episode of laughter as the initial symptom of an ischemic stroke].

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BACKGROUND Pathological laughter occurs in pseudobulbar paralysis, in psychiatric disorders and as a sign of convulsions (gelastic crisis). An extremely rare form is the "fou rire prodromique' a pathological episode of laughter preceding a stroke. The pathogenesis is unknown and classically

Isolated facial palsy: a new lacunar syndrome.

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Three cases of sudden isolated upper motor neuron facial palsy and two with associated pseudobulbar palsy have been seen. All were without significant limb weakness. Computed tomography demonstrated small deep infarcts in the internal capsular/corona radiata regions. Pure upper motor neuron facial

Focal increased 18F FP-CIT uptake in a recent ischemic lesion in the frontal lobe.

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A 65-year-old male patient was referred for rapid functional decline over 1 month with dysphagia and dysarthria. Past history disclosed left side weakness for 5 years. F FP-CIT PET/CT was performed to evaluate the possibility of pseudobulbar palsy. Images showed a defect in the right posterior

Brachial diplegia in central pontine myelinolysis.

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A patient developed weakness in the upper limbs, eventually causing brachial diplegia with only slight paresis of the legs after rapid correction of severe hyponatraemia. Pseudobulbar palsy, mental confusion and urinary incontinence were also present. CT scan showed a zone of lucency in the pons.

[A 59-year-old woman with personality change and abnormal behavior followed by amyotrophy and dementia].

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We report a 59-year-old woman with generalized amyotrophy and dementia. She showed personality change at 53 years of age. When she was 56 years old, she began to show abnormal and violent behaviors. At age 58, she developed dysphagia and amyotrophy of upper limbs. She was admitted to a hospital for

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