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adrenocortical hyperfunction/edém
Odkaz je uložen do schránky
Strana 1 z 37 Výsledek
Cyclical edema and hypokalemia due to occult episodic hypercorticism.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
Yearly episodes of edema, hypokalemia, anxiety, and depression were found to be due to cortisol and deoxycorticosterone surges secondary to a pituitary adenoma in a woman without any of the usual clinical features of Cushing's syndrome. During the long clinical remissions, she had no recognizable
[Hypercorticism due to adrenal adenoma with glucorticoid and androgen hypersecretion, edema and hypokalemia, severe metabolic disorders].
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
Endogenous hypercortisolism inducing reversible ocular hypertension.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
Pulmonary artery thrombosis in three dogs with hyperadrenocorticism.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
Varying degrees of respiratory distress developed in 3 dogs in which hyperadrenocorticism was diagnosed. The respiratory distress was attributed to pulmonary artery thrombosis. Radiography revealed pleural effusion, increased diameter and blunting of the pulmonary arteries, lack of perfusion of the
[Diabetes mellitus and massive lower leg edema without heart failure].
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
BACKGROUND
The 47-year-old male patient was admitted to the hospital because of newly diagnosed diabetes and elevated liver function tests (gamma glutamyl transferase 303 U/l). On admission the patient reported a reduction of appetite, which had increased during the past 2 weeks, fatigue, muscular
ISLET-CELL CARCINOMA (ZOLLINGER-ELLISON SYNDROME) WITH FULMINATING ADRENOCORTICAL HYPERFUNCTION AND HYPOKALEMIA.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
The production of ACTH-like material by tumours arising in non-endocrine tissue may initiate severe adrenocortical hyperfunction. The pathogenesis and clinical and laboratory features of Cushing's syndrome associated with such tumours are characteristic. The autonomous production by the tumour of
Significant hypercortisolism during fractionated radiotherapy in a patient with a large corticotroph adenoma: a case report and literature review.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
OBJECTIVE
We describe a patient with a large, invasive corticotroph adenoma who developed severe hypercortisolism shortly after starting fractionated radiotherapy.
METHODS
We reviewed the patient's clinical course, along with relevant literature for similar reported cases.
RESULTS
A 29-year-old man
Hyperadrenocorticism of calorie restriction contributes to its anti-inflammatory action in mice.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
Calorie restriction (CR), which lengthens lifespan in many species, is associated with moderate hyperadrenocorticism and attenuated inflammation. Given the anti-inflammatory action of glucocorticoids, we tested the hypothesis that the hyperadrenocorticism of CR contributes to its attenuated
Hyperadrenocorticism, attenuated inflammation, and the life-prolonging action of food restriction in mice.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
Food restriction (FR), which extends life span, is associated with an enhanced diurnal elevation of glucocorticoids. This increase in glucocorticoids may contribute to longevity by chronically enhancing the same protective mechanisms mobilized during acute stress. The objective of this study was to
Cushing's disease cycling over ten years.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
Cycles of excessive cortisol secretion have been reported interposed with phases of remission lasting from few days to several months. In this study we report a ten year follow up on a patient who had four episodes of hypercortisolism and Cushing's syndrome associated with hypokalemia and edema and
Cyclic Cushing's disease with paradoxical response to dexamethasone.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
Cyclic Cushing's disease is an unusual disorder characterised by ACTH-dependent periodical increase of serum cortisol levels, clinically accompanied by peripheral edema, abnormalities of cardiac rhythm and hypokalemia. The condition may be unrecognised for years, since the typical features of
Enhanced Na+/H+ exchange in Cushing's syndrome reflects functional hypermineralocorticoidism.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
BACKGROUND
Patients with Cushing's syndrome exhibit a bimodal distribution of maximal rates of the erythrocyte amiloride-sensitive Na+/H+ exchange (NHE). Enhanced erythrocyte NHE has recently been found in patients with primary aldosteronism.
OBJECTIVE
To test the hypothesis that occult
[Reversible metabolic syndrome].
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
A 52-year-old male patient was admitted to the emergency department with dyspnea and hypertensive urgency. During the previous 6 months, the patient had noticed leg edema, weight gain (particularly in the face and abdomen), and impotence. 1.5 years ago, he was diagnosed with hypertension resistant
Cushing's syndrome patient who exhibited congestive heart failure.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
Cushing's Syndrome (CS) may sometimes lead to dilated cardiomyopathy, even though this condition can be partially or completely reversed after treatment. In this article we report the case of a 28-yr-old woman with CS secondary to adrenal adenoma who exhibited congestive heart failure as an initial
A patient with ectopic cortisol production derived from malignant testicular masses.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
BACKGROUND
A 65-year-old man presented to an oncology clinic with bilateral testicular masses, lower extremity edema, and cushingoid appearance.
METHODS
Measurements of serum cortisol and adrenocorticotropic hormone levels, testicular ultrasound and abdominal CT scans, and review of histopathology
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