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polymyositis/protease
Odkaz je uložen do schránky
14 Výsledek
Membrane defects in Duchenne dystrophy: protease affecting sarcoplasmic reticulum.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
Human muscle sarcoplasmic reticulum (SR) yields three major protein bands. The percent distribution of the mean values of the bands from 15 normal human muscles was 55.4, 14.6, and 30.0 for the 100, 55, and 45-kDa mass proteins, respectively. A mean distribution similar to that in normal muscle SR
Autoantibodies to calpastatin (an endogenous inhibitor for calcium-dependent neutral protease, calpain) in systemic rheumatic diseases.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
We identified an autoantibody that reacts with calpastatin [an inhibitor protein of the calcium-dependent neutral protease calpain (EC 3.4.22.17)]. In early immunoblot studies, sera from patients with rheumatoid arthritis (RA) recognized unidentified 60-, 45-, and 75-kDa proteins in HeLa cell
Giant cell polymyositis and myocarditis associated with myasthenia gravis and thymoma.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
We describe an unusual case of myasthenia gravis. Our patient had been diagnosed as having myasthenia gravis with thymoma at the age of 64 years, and died of acute respiratory failure at the age of 80 years. Post mortem examination revealed CD8-positive lymphocytic infiltration with numerous giant
Proteasome expression in the skeletal muscles of patients with muscular dystrophy.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
Previous investigators have suggested that proteolysis by calpain, a Ca2+-dependent protease, causes muscle fiber degradation in Duchenne and Becker muscular dystrophies (DMD/BMD). Recent evidence indicates that the nonlysosomal ATP-ubiquitin-dependent proteolytic complex (proteasomes) participates
Cellular distribution of proteolytic enzymes in the skeletal muscle of sarcoid myopathy.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
To clarify the mechanism of muscle fiber destruction in sarcoid myopathy, muscle biopsy specimens were examined from patients with sarcoid myopathy, polymyositis, or dermatomyositis. In sarcoid myopathy, noncaseating granulomatous lesions were located in the perimysium or endomysium or both. Little
ADAM-17 is expressed in the inflammatory myopathy and is involved with interstitial lung disease.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
The "A disintegrin and metalloprotease" (ADAM) family is thought to play an important role in tissue destruction and inflammatory reactions. ADAM-17 was first described as the protease responsible for tumor necrosis factor (TNF)-α shedding. Here, we have shown the expression of ADAM-17 in
Proteolytic enzyme activities in mononuclear cells and granulocytes of patients with various neurological disorders.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
Our studies showed a significantly increased neutral protease activity in mononuclear cells in patients with MS in relapse, active neuro-Behçet's disease, acute disseminated encephalomyelitis (ADEM) and polymyositis. Furthermore, in patients with ADEM there was a significant increase in the
Proteolytic enzyme activities of macrophages and lymphocytes in neurological diseases.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
Studies showed a significant decrease in the macrophage neutral protease and lymphocyte acid protease activities in patients with multiple sclerosis in remission, a significantly decreased neutral protease activity in macrophages in patients with myasthenia gravis and a significantly decreased acid
Chemokine-like factor expression in the idiopathic inflammatory myopathies.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
OBJECTIVE
We evaluated the expression of chemokine-like factor (CKLF) in biopsied muscle fibers in inflammatory myopathies, non-inflammatory myopathies and neurologically diseased controls.
METHODS
We studied the expression of CKLF in 15 polymyositis (PM), five dermatomyositis (DM), 15
Inducible overexpression of wild-type prion protein in the muscles leads to a primary myopathy in transgenic mice.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
The prion protein (PrP) level in muscle has been reported to be elevated in patients with inclusion-body myositis, polymyositis, dermatomyositis, and neurogenic muscle atrophy, but it is not clear whether the elevated PrP accumulation in the muscles is sufficient to cause muscle diseases. We have
Abnormal glycosylation of alpha 2-macroglobulin, a non-acute-phase protein in patients with autoimmune diseases.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
Previous studies from this and other laboratories have shown that abnormal glycosylation of several acute-phase proteins can be detected in various pathological conditions including autoimmune diseases. In the present study, we have investigated if abnormal glycosylation is limited to acute-phase
Lysosomal and nonlysosomal hydrolases of skeletal muscle in neuromuscular diseases.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
The activities of four lysosomal and two nonlysosomal hydrolases were studied in skeletal muscle biopsy samples from patients with neuromuscular diseases and from controls. beta-Glucosaminidase activity was increased in polymyositis. beta-Glucuronidase and alkaline protease activities were elevated
Pegylated interferon induced myasthenia crisis--a case report.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
Interferons (IFNs) have antiviral, antimitogenic, and immunostimulatory effects and are often used in the treatment of viral hepatitis and some neoplasms. Combination pegylated IFN-alpha and ribavirin therapy is currently recommended for the treatment of hepatitis C. Triple therapy, with the
Calpain inhibition attenuates intracellular changes in muscle cells in response to extracellular inflammatory stimulation.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
Idiopathic inflammatory myopathies (IIMs), comprising of polymyositis, dermatomyositis, and inclusion-body myositis, are characterized by muscle weakness and various types of inflammatory changes in muscle cells. They also show non-inflammatory changes, including perifascicular atrophy,
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