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hemifacial spasm/asthenia
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Facial Weakness, Otalgia, and Hemifacial Spasm: A Novel Neurological Syndrome in a Case-Series of 3 Patients With Rheumatic Disease.
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Bell palsy occurs in different rheumatic diseases, causes hemifacial weakness, and targets the motor branch of the 7th cranial nerve. Severe, persistent, and refractory otalgia having features of neuropathic pain (ie, burning and allodynic) does not characteristically occur with Bell palsy. Whereas
Chemomyectomy of the orbicularis oculi muscles for the treatment of localized hemifacial spasm.
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OBJECTIVE
To report our experience with doxorubicin chemomyectomy as an alternative to other treatments for hemifacial spasm (HFS).
METHODS
A prospective, open study Phase I clinical trial of chemomyectomy.
METHODS
A hospital-based, referral neuro-ophthalmology and oculoplastic
Multiple Sclerosis Presenting with Facial Twitching (Myokymia and Hemifacial Spasms).
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Multiple sclerosis (MS) is a chronic inflammatory demyelinating disease of the central nervous system. The etiology is insufficiently understood. Autoimmune, genetic, viral, and environmental factors have been hypothesized. MS is twice as common in women as in men between the ages of 20 and 50
Aneurysmal expansion presenting as facial weakness: case report and review of the literature.
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OBJECTIVE
Facial paralysis and hemifacial spasm are rare presentations of aneurysms in the posterior fossa. We report an unusual case of rapidly progressive facial palsy caused by the acute expansion of an arteriovenous malformation-associated anteroinferior cerebellar artery aneurysm. The case is
Hemifacial spasm caused by the brainstem developmental venous anomaly: A case report and review of the literature
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Background: Hemifacial spasm (HFS) is usually caused by vascular compression of the root exit zone (REZ) of the facial nerve. Dual compression of the REZ by veins and arteries is also associated with HFS, but venous origin alone is rarely
Surgical Management of Hemifacial Spasm Associated with Chiari I Malformation: Analysis of 28 Cases.
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OBJECTIVE
Hemifacial spasm (HFS) associated with Chiari I malformation (CIM) is rare. This study aimed to analyze the frequency of HFS associated with CIM in our department and further to investigate the clinical characteristics, treatment strategies, and outcomes of these
Prevalence of Bruxism in Hemifacial-Spasm Patients.
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OBJECTIVE
A previous study reported an increased prevalence of bruxism (25%) in patients with cranio-cervical dystonia (CCD) compared to normal controls (13%). CCD can affect the muscles of the head and neck. Besides the CCD affecting these muscles, hemifacial spasm (HFS) is a form of peripheral
Facial nerve injury and hemifacial spasm.
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We studied evidence of facial nerve damage in patients with hemifacial spasm. Three types of evidence of nerve damage were analyzed: objectively measured weakness in eyelid protractor strength, clinically evident weakness of muscles innervated by the seventh nerve, and clinically evident aberrant
A case of hemifacial spasm caused by an artery passing through the facial nerve.
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Hemifacial spasm (HFS) is a clinical syndrome characterized by unilateral facial nerve dysfunction. The usual cause involves vascular compression of the seventh cranial nerve, but compression by an artery passing through the facial nerve is very unusual. A 20-year-old man presented with left facial
[Long-term observation on two types of surgical operations for idiopathic hemifacial spasm].
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OBJECTIVE
To evaluate and compare the long-term curative effects of two types of surgical operations, microvascular decompression (MVD) and microneurovascular decompression neurocombing neurotraction draw (MVDCTD) for idiopathic hemifacial spasm (IHFS).
METHODS
Five hundred and fifty-four patients
Systematic Literature Review of AbobotulinumtoxinA in Clinical Trials for Blepharospasm and Hemifacial Spasm.
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BACKGROUND
The aim was to elucidate clinical trial efficacy, safety, and dosing practices of abobotulinumtoxinA (ABO) treatment in adult patients with blepharospasm and hemifacial spasm. To date, most literature reviews for blepharospasm and hemifacial spasm have examined the effectiveness of all
Hemifacial spasm: treatment by microvascular decompression of the facial nerve at the pons.
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Hemifacial spasm is a syndrome of intermittent and tonic unilateral facial muscular contraction; mild facial weakness on the same side is also frequently present. Hemifacial spasm can be differentiated clinically from habit spasms, blepharospasm, facial synkinesis following Bell's palsy, facial
Microvascular decompression by the retrosigmoid approach for idiopathic hemifacial spasm: experience with 300 cases.
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Three hundred patients with idiopathic hemifacial spasm who underwent microvascular decompression through the retrosigmoid approach are reported. Vascular compression was found in every patient on operation. The results of 1 to 6 years of follow-up show that 276 patients are free of the symptom, 4
Hemifacial spasm: clinical characteristics of 321 Indian patients.
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Hemifacial spasm (HFS) is a common neurological disorder characterized by involuntary tonic and clonic contractions of the muscles innervated by the facial nerve. We aimed to describe clinical features, common antecedents, triggers and relieving factors in patients with hemifacial spasm to study the
[CP angle meningioma presenting as hemifacial spasm].
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An interesting case of CP angle meningioma presenting with hemifacial spasm is reported. The patient was a 64-year-old woman with left hemifacial spasm of 18 years' duration which was the initial and the only symptom she had had for ten years until other signs and symptoms including tinnitus and
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