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isonicotinic acid/epileptisk anfald
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Effect of isonicotinic acid hydrazide on extracellular amino acids and convulsions in the rat: reversal of neurochemical and behavioural deficits by sodium valproate.
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We have studied the effect of isonicotinic acid hydrazide (INH), a convulsant agent, on the extracellular levels of amino acids in the hippocampus, and the effect of sodium valproate (VPA) administration in INH-treated rats. INH (250 mg/kg) caused a rapid and sustained decrease in basal levels of
Characterization of the anticonvulsant profile of isonicotinic acid benzylamide in various experimental seizure models in mice.
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This study focused on the evaluation of anticonvulsant properties of isonicotinic acid benzylamide (iso-Nic-BZA) in numerous experimental seizure models (maximal electroshock [MES]-, bicuculline [BIC]-, pentylenetetrazole [PTZ]-, pilocarpine [PILO]-,
A correlation between changes in GABA metabolism and isonicotinic acid hydrazide-induced seizures.
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[Changes in the neurosecretory system of rabbits during convulsions induced by isonicotinic acid hydrazide].
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[Effect of ACTH and some adrenocortical hormones on convulsions induced by isonicotinic acid hydrazide].
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The effect of hypoxia on isonicotinic acid hydrazide-induced seizures in chicks during ontogenesis.
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[Explication of antagonistic action of certain drugs toward preparations of isonicotinic acid hydrazide group producing convulsions].
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Changes in the neurosecretory system of the rabbit in the course of convulsions caused by isonicotinic acid hydrazide.
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Effects of pyridoxine, gamma-aminobutyric acid and gamma-amino-beta-hydroxybutyric acid on the convulsions of dogs caused by toxopyrimidine or isonicotinic acid hydrazide.
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Effect of trimethyltin on chemically-induced seizures.
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The effect of trimethyltin (TMT; 4.26 mg/kg i.p.), at 1 and 14 h following administration, on chemically-induced seizures in mice is reported. At 1 h following administration, TMT decreased seizure responsiveness and protected animals from bicuculline, isonicotinic acid hydrazide (INH) and
Assessment of the anticonvulsant potency of various benzylamide derivatives in the mouse maximal electroshock-induced seizure threshold model.
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OBJECTIVE
The aim of this study was to assess the anticonvulsant potency of 6 various benzylamide derivatives [i.e., nicotinic acid benzylamide (Nic-BZA), picolinic acid 2-fluoro-benzylamide (2F-Pic-BZA), picolinic acid benzylamide (Pic-BZA), (RS)-methyl-alanine-benzylamide (Me-Ala-BZA),
Anticonvulsant activity of the glial GABA uptake inhibitor, THAO, in chemical seizures.
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The antiseizure activity of the glia-selective GABA uptake inhibitor, 5,6,7,8-tetrahydro-4H-isoxazolo[4,5-c]azepin-3-ol (THAO), was evaluated in rats in models of acute chemoconvulsion. In these experiments, intracerebroventricular administration of the drug 30 min prior to testing in doses between
Anticonvulsant activity of intracerebroventricularly administered glial GABA uptake inhibitors and other GABAmimetics in chemical seizure models.
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The antiseizure activities of glial or neuronal GABA uptake inhibitors and GABA agonists were compared following intracerebroventricular administration in 2 acute models of chemoconvulsion in rats. The glia-selective GABA uptake inhibitor, 4,5,6,7-tetrahydroisoxazolo[4,5-c]pyridin-3-ol (THPO), given
Effects of cannabidiol on behavioral seizures caused by convulsant drugs or current in mice.
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In mice, running, clonic and tonic convulsions and lethality were assessed following transcorneal (electroshock) current or convulsant drugs, each administered alone and after cannabidiol (CBD) pretreatment. CBD prevented tonic convulsions caused by a convulsant current (CC) 99.99, and by the
Genetic analysis of susceptibility to isoniazid-induced seizures in mice.
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Four sets of recombinant inbred lines of mice have been used to analyze genetic differences in acute toxicity of the drug, isonicotinic acid hydrazide. Standard inbred strains, their F(1) hybrids and recombinant inbred strains were all challenged with a single dose of the drug. The percent mortality
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