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lichen planus/proline
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3 resultater
Codon72 polymorphism in the p53 tumor suppressor gene in oral lichen planus lesions in a Thai population.
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BACKGROUND
Oral lichen planus (OLP) is a T-cell mediated autoimmune disease in which autocytotoxic CD8+ T cells (CTLs) trigger apoptosis of oral epithelial cells. Activated CTLs can produce Fas ligand and by binding to Fas lead to apoptosis. This Fas pathway and the action of p53 tumour suppressor
Polymorphism of tumor protein p53 codon 72 showed no association with oral lichen planus in Shiraz, Iran.
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BACKGROUND
Oral lichen planus (OLP) is a chronic relapsing inflammatory disease involving T-lymphocyte aggression targeted to the basal layer of the oral mucosa. The tumor suppressor tumor protein p53 (TP53), located on chromosome 17, has been involved in various malignancies.
OBJECTIVE
This study
Evaluation of polymorphism of P53 protein codon 72 in oral lichen planus by PCR technique.
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Background. Our research was aimed to study p53 protein codon 72 polymorphism, a single base pair change of either arginine (Arg; CGC) or proline (Pro; CCC) that creates 3 distinct genotypes in reticular oral lichen planus (OLP) in comparison to oral SCC which is the most common oral
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