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lumicolchicine/ødem
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Dopamine restores lung ability to clear edema in rats exposed to hyperoxia.
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Exposure to hyperoxia causes lung injury, decreases active sodium transport and lung edema clearance in rats. Dopamine (DA) increases lung edema clearance by stimulating vectorial Na+ flux and Na, K-ATPase function in rat alveolar epithelium. This study was designed to test whether DA (10(-)5 M)
Catecholamines increase lung edema clearance in rats with increased left atrial pressure.
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During hydrostatic pulmonary edema, active Na(+) transport and alveolar fluid reabsorption are decreased. Dopamine (DA) and isoproterenol (ISO) have been shown to increase active Na(+) transport in rat lungs by upregulating Na(+)-K(+)-ATPase in the alveolar epithelium. We studied the effects of DA
Isoproterenol improves ability of lung to clear edema in rats exposed to hyperoxia.
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Exposure of adult rats to 100% O(2) results in lung injury and decreases active sodium transport and lung edema clearance. It has been reported that beta-adrenergic agonists increase lung edema clearance in normal rat lungs by upregulating alveolar epithelial Na(+)-K(+)-ATPase function. This study
beta-adrenergic stimulation restores rat lung ability to clear edema in ventilator-associated lung injury.
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Mechanical ventilation with high tidal volume (HVT) causes lung injury and decreases the lung's ability to clear edema in rats. beta-adrenergic agonists increase active Na(+) transport and lung edema clearance in normal rat lungs by stimulating apical Na(+) channels and basolateral Na,K-ATPase in
Effect of phenylephrine on alveolar fluid clearance in ventilator-induced lung injury.
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OBJECTIVE
To investigate the effect of phenylephrine (an α-adrenergic agonist) on alveolar fluid clearance (AFC) in ventilator-induced lung injury and the possible mechanism involved.
METHODS
A total of 170 male Wistar rats were randomly allocated into 17 groups (n=10) using random number tables.
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