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pseudotumor cerebri/ødem
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Bilateral optic disk edema caused by sarcoidosis mimicking pseudotumor cerebri.
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OBJECTIVE
To present a case of retrobulbar optic nerve and chiasm sarcoidosis that mimicked pseudotumor cerebri.
METHODS
A 34-year-old, thin, black woman presented with transient visual obscurations, normal visual acuity, bilateral optic disk edema, and enlarged blind spots. Clinical, medical, and
Unilateral high myopia leading to asymmetric disc edema in idiopathic intracranial hypertension
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Asymmetrical disc edema in idiopathic intracranial hypertension (IIH) is an uncommon finding, with an unclear understanding. This report reinforces the importance of recognizing this entity in IIH diagnosis, and not misdiagnose the condition as unilateral disc edema. In this unique report, the
Classification of the cerebral edemas with reference to hydrocephalus and pseudotumor cerebri.
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Cerebral edema is a common clinical disorder that results from an abnormal increase in water content within the extracellular (EC) compartment of the brain. It is distinguished from two other types of brain bulk enlargement: (1) vascular swelling, caused by arterial dilatation or venous obstruction;
Endolymphatic hydrops in idiopathic intracranial hypertension: prevalence and clinical outcome after lumbar puncture. Preliminary data.
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Idiopathic intracranial hypertension is characterized by raised intracranial pressure (ICP) without any underlying pathology, presenting with (IIH) or without papilledema (IIHWOP). Headache, often on daily basis, is the most frequent symptom. Among audiovestibular symptoms, tinnitus and dizziness
Idiopathic intracranial hypertension and orthostatic edema may share a common pathogenesis.
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Our aim was to determine the frequency of orthostatic edema (OE) in patients with idiopathic intracranial hypertension (IIH). We evaluated 30 women with IIH for evidence of OE by comparing sodium and water excretion in the recumbent and standing postures and morning and evening body weights. Data
Idiopathic intracranial hypertension. Lack of histologic evidence for cerebral edema.
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OBJECTIVE
To study brain histologic features in two cases of idiopathic intracranial hypertension (pseudotumor cerebri) at autopsy.
METHODS
Formaldehyde solution-fixed sections of cerebral white matter in two cases and structures of the sensory visual system in one case were
[Unexplained bilateral optic disk edema. So-called idiopathic intracranial hypertension of drug origin should be suspected].
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In 2 clinical cases of bilateral optic disk oedema, the optic disk oedema was part of a so-called benign, or rather idiopathic, intracranial hypertension. Aetiological investigations were unable to detect any intracranial or systemic anomaly, except the fact that both patients had been on a
Irreversibility of transverse venous sinus stenosis and optic nerve edema post-lumbar puncture in idiopathic intracranial hypertension.
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[Acute nonbacterial meningitis with papillary edema--pathogenetic observation of benign intracranial hypertension].
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[Pseudotumor cerebri and unilateral papillary edema. Presentation of a case].
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MRI of optic disc edema in childhood idiopathic intracranial hypertension.
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[The apallic syndrome in brain edema caused by pseudotumor cerebri].
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[Benign intracranial hypertension. A clinical, pathophysiological and diagnostic study (author's transl)].
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Sixteen unpublished observations of Benign intracranial hypertension were reviewed from a clinical, aetiological and prognostic standpoint. The hypothesis that this affection could be caused by some disturbance of the C.S.F. resorption was assessed using an experimental tests battery allowing the
Disk edema subsequent to renal transplantation.
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A 12-year-old child was discovered (following successful renal transplantation) to have asymptomatic bilateral disk edema with preservation of optic nerve function. He had been maintained on cyclosporine, imuran and prednisone. Evaluation revealed normal intracranial imaging and normal CSF though
A Clinical Study of Optic Disc Edema in a Tertiary Eye Center of Nepal
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Purpose: To evaluate the etiology and clinical presentation of cases with optic disc edema presenting to a tertiary eye center of Nepal.
Background: The etiology of optic disc
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