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Seite 1 von 123 Ergebnisse
The similarity in action of hypoxia and platelet-activating factor on smooth muscle cells of coronary arteries: possible explanation for hypoxic coronary spasm development.
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Studies in isolated smooth muscles and single cells of coronary arteries have demonstrated that both hypoxia and platelet activating factor (PAF) in a similar manner increased contractile force and Ca-activated K currents. The specific antagonists of PAF receptors BN 52021 and WEB 2886 significantly
[Various ECG and metabolic indices during experimental coronary spasm in rats adapted to hypoxia and hypercapnia].
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Hypoxia and spasms in the cerebral vasculature.
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Genetic study of infantile spasm with hypsarrhythmia.
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Infantile spasms (IS) appear to be a distinct syndrome complicated by heterogeneous etiology. Family data support a multifactorial model involving a polygenic determination of susceptibility to IS but requiring environmental factors such as anoxia, birth trauma, or immunization to precipitate
Evaluation of Corneal Morphology in Patients With Hemifacial Spasm.
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[Morphological changes in the fetal part of the allantoic placenta in rats exposed to acute hypoxia].
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The aim of this study was to analyze the response of cyto- and syncytiotrophoblast elements of fetal part of allantoic placenta to general acute hypoxia and to determine their morphologic characteristics, which provide the basis for structural changes in placenta and for the disturbances in
Contribution of hypoxia to the development of cardiomyopathy in hamsters.
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OBJECTIVE
It has been hypothesized that microvascular spasms cause cardiomyopathy. To elucidate the contribution of hypoxia to the development of cardiomyopathy, the newly-developed hypoxia tracer, Tc-99m nitroimidazole, was applied to detect myocardial hypoxia in a hamster model.
METHODS
Tc-99m
Risk factors of infantile spasms compared with other seizures in children under 2 years of age.
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To analyze the magnitude of the risk factors for infantile spasms, we evaluated the records of 80 children with infantile spasms, 474 children with other types of epilepsy, 2,196 children with febrile seizures, and 262 children with CNS infections. There was a family history of seizures in 13.8% of
Early clinical and EEG features of infantile spasms in Down syndrome.
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The combination of West syndrome (WS) and Down syndrome appears not to be coincidental. Fourteen patients free of cardiac malformation or history of perinatal hypoxia were referred and investigated before they had received any treatment and were followed to the mean age of 4.5 years (range 19 months
[Coronary spasm and diffuse coronary vasoconstriction responsible for transient left ventricular insufficiency].
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A 52 year old patient presenting with spontaneous anginal chest pain for 4 days was admitted to hospital for a more intense and prolonged chest pain associated with signs of left ventricular failure (gallop, pulmonary crepitations, hypoxemia). Coronary angiography showed marked septal hypokinesia
Spasm of the coronary arteries: causes and consequences (the scientist's viewpoint).
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Both beta 1- and alpha 1-adrenoceptors are present on canine coronary arteries, and they are accessible to norepinephrine released from the sympathetic nerves. Under normal conditions, these arteries relax because of the predominance of the beta 1-adrenoceptors, whereas constriction prevails in the
[Hypoxia of the central nervous system in rats during acceleration brain injury].
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The authors present the results of investigations of the effects of acceleration craniocerebral injury in rats on PaO2 and resulting central nervous system hypoxia. PaO2 fall by 30% of the control value occurred in 25% of rats and in the group of most severe injury this fall amounted to 36% already
Mechanism and treatment principle for cerebral vessel spasm caused by concussion.
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OBJECTIVE
To discuss the mechanism of cerebral vessel spasm caused by concussion and the effect of Nimodipine on concussion.
METHODS
A total of 224 patients who were treated from March 1995 to October 1999 were divided into two groups randomly, ie, Nimodipine group (113 cases) and control group (111
[Vascular spasms in microsurgery].
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Human arteries and veins contract with hypoxia and deprivation of substrate provoked by increasing calcium inflow into the cell and reduced energy. Such spasm may be eliminated by phosphoenolpyruvate which loads up the cell's energy, blocking glycogen reduction at the same time. Reperfusion will
[Recording muscle spasm in the musculus erector spinae using in vivo 31P magnetic resonance spectroscopy in patients with chronic lumbalgia and generalized tendomyopathies].
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Using 31P nuclear magnetic resonance, the following parameters were determined in the resting musculus erector spinae of five patients suffering from chronic low back pain, five patients with fibromyalgia, and five healthy controls: Inorganic phosphate (Pi), phosphocreatine (PCr), ATP gamma, ATP
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