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Clinical significance of the apical late systolic ejection murmur: a new phonocardiographic sign indicating dynamic mid-left ventricular obstruction.

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Systolic ejection murmurs of the left heart usually have their peak during early to mid-systole. Few reports have addressed ejection murmurs with their peak at late systole. We evaluated the clinical significance of an apical systolic ejection murmur with a peak intensity during late systole using

Intracardiac phonocardiography in experimental left ventricular cavity obliteration: potential clinical applicability for the distinction of obliterating left ventricle from hypertrophic obstructive cardiomyopathy.

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Intracardiac sound was measured in six dogs, four with left ventricular cavity obliteration induced by isoproterenol, and two with catheter entrapment. In left ventricular cavity obliteration, no murmur occurred within the left ventricle. Whenever a systolic murmur occurred, it was distal to the

Left Ventricular Mid-Cavity Hypertrophic Cardiomyopathy with Myeloproliferative Neoplasm Leading to Multiple Cerebral Emboli.

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Here we present a case of 55 year old male who presented with lower respiratory tract infection and clinical findings of systolic murmur at apex and hepatosplenomegaly and later on multiple cerebral emboli which on further evaluation turned out to be myeloproliferative neoplasm associated with

[Echocardiographic features of false tendons: with special reference to phonocardiographic significance (author's transl)].

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Echocardiographic features consistent with the findings of false tendons (FTs) were described in five out of 1,000 consecutive cases, and they were studied in order to determine whether FT was responsible for the systolic murmurs. Three had heart diseases including aortic regurgitation, 3 degrees

[Obstructive cardiomyopathy and stenosing coronary atherosclerosis. Apropos of 5 cases].

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Five cases of hypertrophic obstructive cardiomyopathy with coronary artery disease are reported; 2 males and 5 females, aged 27, 49, 64, 67, and 70 years respectively. Four patients had angina, the other being asymptomatic. The diagnosis of hypertrophic obstructive cardiomyopathy was based on the

[Cardiac manifestation of the mucopolysaccharidoses: periodical echocardiographic evaluation in six cases].

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Serial non-invasive studies including echocardiography were performed for the evaluation and follow-up of the cardiac lesions in six cases with genetic mucopolysaccharidoses. These cases were classified by the enzyme assay into one case of Scheie syndrome, three of Hunter syndrome, one of Sanfilippo

[Cross-sectional echocardiographic findings of a case with ruptured aneurysm of the sinus of Valsalva into the right atrium (author's transl)].

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A case with ruptured aneurysm of the sinus of Valsalva into the right atrium directly visualized by cross-sectional echocardiography was reported. A 31-year-old male was referred to our hospital for evaluation of recently developed cardiac murmur. A systolic ejection murmur and diastolic blowing

Right ventricular endomyocardial fibrosis. Diagnosis and management.

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The clinical course, noninvasive and invasive diagnostic findings in 4 patients (pt) with endomyocardial fibrosis are reported. All patients (16-50 years) were in functional class III-IV (NYHA). Central venous pressure with large a- and v-waves was elevated in all; liver enlargement and peripheral

[Sigmoid septum causing left ventricular outflow tract obstruction: a case report].

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A 67-year-old man with a sigmoid septum causing the left ventricular outflow obstruction by inotropic stimulation was reported. This patient was admitted to the Hospital of the University of Tsukuba because of chest pain. Phonocardiography revealed a systolic ejection murmur which was intensified by

[Unruptured aneurysms of the sinus of Valsalva disclosed by syncopal disorders of cardiac excitability. Apropos of 2 cases].

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Aneurysms of the sinus of Valsalva are rarely diagnosed before rupture into the cardiac cavities which usually leads to the appearance of a continuous murmur and cardiac failure. In the two cases described, the presenting symptom of the aneurysm was syncope due to cardiac hyperexcitability:

Intracardiac sound as a diagnostic adjunct in subaortic stenosis.

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The purpose of this investigation is to demonstrate the potential diagnostic value of intracardiac sound recordings in patients with subaortic stenosis. Intracardiac pressure and sound were measured in 10 patients with various types of subaortic obstructions using a catheter-tip micromanometer.

[The incidence and clinical significance of the echocardiographic finding of false chordae tendineae].

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The presence of false tendons (FT) with M-mode and 2-D echocardiography in a consecutive series of 916 patients (520 men--396 women) with a wide age range (9-83 y) was investigated, and a possible correlation with heart systolic murmurs and/or heart arrhythmias evaluated. The problems that FT cause

Can fibrotic bands in the aortic arch cause innocent murmurs in childhood?

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Children with innocent murmurs are often referred to a paediatric cardiologist for diagnosis. The most common murmurs of early childhood are the so-called Still's murmurs, followed by ejection murmurs across the pulmonary or aortic vessels, and the venous hum. There also exists a high coincidence of

[The differential diagnosis of various forms of hypertrophic cardiomyopathy and hypertension with inadequate left ventricular hypertrophy].

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In order to develop the tentative criteria of the differential diagnosis, 18 patients with obstructive hypertrophic cardiomyopathy (OHCMP), 3 with nonobstructive hypertrophic cardiomyopathy (NOHCMP), 8 with essential hypertension (EH) with inadequate left ventricular hypertrophy (LVH) and 10 normal

Detection of left ventricular false tendons by two-dimensional echocardiography.

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In a prospective series of 300 consecutive patients referred for a two-dimensional echocardiogram, the presence of left ventricular false tendons was searched. The diagnosis of a false tendon was made when a clear linear echo was continuously observed within the left ventricular cavity from the

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