ascorbate oxidase/hemorrhage

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Hemoglobin-mediated oxidant damage to the central nervous system requires endogenous ascorbate.

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Hemorrhage within the central nervous system (CNS) may be associated with subsequent development of seizure states or paralysis. Prior investigations indicate that hemoglobin, released from extravasated erythrocytes, may be toxic to the CNS by promoting peroxidation of lipids and inhibition of

Hemoglobin-induced lipid peroxidation in the retina: a possible mechanism for macular degeneration.

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To investigate a possible link between subretinal hemorrhage and macular degeneration, oxyhemoglobin (HbO2) or methemoglobin (metHb) was incubated with retinal homogenate and unsaturated phospholipid peroxidation was monitored by (a) assay of thiobarbituric acid-reactive substances (TBARS), (b)

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