hemianopsia/inflammation

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[Case of neuro-Behçet disease with homonymous quadrantanopsia due to an inflammatory lesion involving the lateral geniculate body].

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We report a rare case of a 57-year-old woman of neuro-Behçet disease with homonymous quadrantanopsia due to an inflammatory lesion involving the lateral geniculate body. She had oral and genital ulcers since 1983, and uveitis since May 1985. She received diagnosis of incomplete Behçet disease and

Tumoral Presentation of Homonymous Hemianopia and Prosopagnosia in Cerebral Amyloid Angiopathy-Related Inflammation.

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While cerebral amyloid angiopathy is a common cause of lobar hemorrhage, rarely it may be associated with an inflammatory response, thought to be incited by amyloid deposits. We report a 73-year-old woman with an extensive cancer history who presented with tumor-like lesions and symptoms of

[Bitemporal hemianopia as presenting sign of severe ethambutol toxicity].

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BACKGROUND Optic neuropathy is a severe and well-known complication of ethambutol treatment. If not detected early, it may lead to profound and irreversible vision loss. METHODS We report the case of a 83-year-old female patient, referred for rapidly progressive, painless, bilateral visual loss,

[A case of systemic lupus erythematosus associated with thrombotic thrombocytopenic purpura manifestating homonymous hemianopsia].

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We report on a 35-year-old Japanese woman with systemic lupus erythematosus (SLE) in whom homonymous hemianopsia developed. In August 1987, SLE was diagnosed. In November 1987, the patient was admitted to our hospital because of severe thrombotic thrombocytopenic purpura; however, clinical symptoms

Cortical dysplasia with angiodysgenesis and chronic inflammation in multifocal partial epilepsy.

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A 25-year-old man with a long history of temporal lobe epilepsy developed right occipital lobe seizures and a progressive right homonymous hemianopia. MRI showed diffuse enhancement of the left temporoparieto-occipital white matter and cortical thickening of the left medial temporal lobe. The

In vivo evolution of biopsy-proven inflammatory demyelination quantified by R2t* mapping.

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A 35-year-old man with an enhancing tumefactive brain lesion underwent biopsy, revealing inflammatory demyelination. We used quantitative Gradient-Recalled-Echo (qGRE) MRI to visualize and measure tissue damage in the lesion. Two weeks after biopsy, qGRE showed significant R2t* reduction in the left

Intracranial Inflammatory Pseudotumors Associated with Immunoglobulin G4-Related Disease Mimicking Multiple Meningiomas: A Case Report and Review of the Literature.

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BACKGROUND Immunoglobulin G (IgG)4-related disease is a syndrome that forms inflammatory pseudotumors with increasing IgG4-positive plasma cells and lymphocytes infiltrating the exocrine gland and other organs. The concept of this disease gradually has gained more recognition. However, reports of

Tumefactive demyelination: an unusual cause of a spontaneously resolving homonymous hemianopia.

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A 21-year-old man presented to eye casualty complaining of altered vision associated with headache and vomiting upon waking. Clinical examination was unremarkable except for a right-sided homonymous hemianopia. The MRI scan of the brain revealed a space occupying lesion within the occipital lobe and

Optic Neuropathy, Secondary to Ethmoiditis, and Onodi Cell Inflammation during Childhood: A Case Report and Review of the Literature.

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Optic neuropathy consists of several etiological events. The primary etiologies of its acute form include optic neuritis, ischemic optic neuropathy, inflammatory (nondemyelinating) disorders, and trauma. Its subacute and chronic forms are most often linked to compressive, toxic, nutritional, or

Neuroimaging appearance of pituitary abscess complicated with close inflammatory lesions--case report.

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A 13-year-old girl with a pituitary abscess complained of continuous headache and bitemporal hemianopsia after a common cold. However, she had no inflammatory reactions on admission. Computed tomography showed a low-density sellar mass lesion extending to the suprasellar cistern with a peripheral

[Homonymous hemianopsia].

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Anatomical principles of the suprachiasmatic pathway. The neuro-ophthalmological examination of homonymous hemianopia: qualitative perimetry (confrontation tests), quantitative perimetry (campimetry on the Bjerrum screen, kinetic perimetry, static perimetry, automatic computer controlled perimetry,

Intrasellar Symptomatic Salivary Gland Rest with Inflammations.

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BACKGROUND Instances of ectopic salivary gland tissue within the pituitary gland are rare, they are mostly asymptomatic, and the underlying pathophysiology of symptomatic cases is unclear. We report a case of intrasellar salivary gland rest that presented clinical symptoms and clearly related to

[Vascular cause of hemianopsia].

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Hemianopsia of vascular origin can be caused by diseases in the carotid circulation (optic tract, proximal third of the optic radiation), as well as in the vertebrobasilar arterial system (middle and distal thirds of the optic tract, striate area/calcarina). Hemianopsia of ischemic origin must be

[Incongruous homonymous quadrantanopia due to suprasellar calcificated lesions].

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A 53 year-old woman with a history of tuberculous meningitis at 3 years of age incidentally found a right incongruous homonymous quadrantanopia in ophthalmologic check-up. On magnetic resonance imaging and helical computed tomography, the left optic tract was obscured by suprasellar calcified

Severe Progressive Multifocal Leukoencephalopathy (PML) and Spontaneous Immune Reconstitution Inflammatory Syndrome (IRIS) in an Immunocompetent Patient.

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Background: Progressive multifocal leukoencephalopathy (PML) is an opportunistic infection with JC-virus (JCV), a papova-virus, affecting mostly oligodendrocytes and the white matter of the central nervous system. Progressive Multifocal Leukoencephalopathy (PML) almost exclusively occurs in

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