hypercapnia/fever

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Elimination of hypercapnia may postpone the clinical presentation of malignant hyperthermia: a case report.

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Malignant hyperthermia is a rare complication in clinical anesthesia, especially associated with the administration of succinylcholine or inhalation anesthetics. A 19-year-old patient, suffering from traumatic mandible fracture, underwent open reduction under general anesthesia. Unfortunately,

[Hypercapnia and mixed acidosis as the only sign of malignant hyperthermia (MH)?].

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METHODS A 34-year-old male (190 cm/100 kg) was scheduled for surgery of the nasal septum. He had had uneventful anaesthesia for appendicectomy 14 years earlier: following 600 mg thiopentone, 180 mg suxamethonium and up to 2 vol.% halothane for 20 min had been used and no symptoms of malignant

Hypercapnia-induced increases in cerebral blood flow do not improve lower body negative pressure tolerance during hyperthermia.

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Heat-related decreases in cerebral perfusion are partly the result of ventilatory-related reductions in arterial CO2 tension. Cerebral perfusion likely contributes to an individual's tolerance to a challenge like lower body negative pressure (LBNP). Thus increasing cerebral perfusion may prolong

[Brain metabolism of certain phosphorus compounds in the first minutes of the aftereffect of hyperthermia and hypercapnia].

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[Combined action of hypoxia, hypercapnia and hyperthermia].

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A Young Man with Accelerated Hyperthermia, Hypercapnia, and Profound Muscle Rigidity after Ingestion of a Weight Loss Agent.

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Preischemic hyperglycemia leads to delayed postischemic hyperthermia.

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OBJECTIVE Temperature alterations are known to influence the outcome of transient ischemia, even when instituted in the postischemic period. Since preischemic hyperglycemia aggravates ischemic brain damage, the question of whether hyperglycemic animals become hyperthermic arose. To explore this

Suspected malignant hyperthermia during sevoflurane anesthesia.

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Malignant hyperthermia is a rare anesthetic-related disorder. We present a case with unusual presentation. A boy aged 3 years and 9 months who was scheduled for Hotz's operation presented normally before the operation. Anesthesia was induced by atropine, thiopental and sevoflurane. Trachea

No effect of skin temperature on human ventilation response to hypercapnia during light exercise with a normothermic core temperature.

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Hyperthermia potentiates the influence of CO(2) on pulmonary ventilation (.V(E)). It remains to be resolved how skin and core temperatures contribute to the elevated exercise ventilation response to CO(2). This study was conducted to assess the influences of mean skin temperature (_T(SK)) and

Prenatal cigarette smoke exposure and postnatal respiratory responses to hypoxia and hypercapnia.

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Prenatal cigarette smoke (CS) exposure, in combination with hypoxia and/or hyperthermia can lead to gasping and attenuated recovery from hypoxia in 7 days old rat pups. We studied 95 unanesthetized spontaneously breathing 14 days old rat pups to investigate if the destabilizing effects of increased

Malignant Hyperthermia: An Anesthesiology Simulation Case for Early Anesthesia Providers.

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The patient is a 40-kg, 12-year-old Caucasian male with history of asthma who is undergoing an elective inguinal hernia repair. There is no family history of anesthesia-related complications. The surgery proceeds under general anesthesia with an IV induction with propofol, fentanyl,

Malignant hyperthermia involving the administration of desflurane.

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OBJECTIVE This report describes an episode of malignant hyperthermia (MH) in a ten year old boy receiving desflurane anaesthesia. METHODS Following induction of general endotracheal anaesthesia with thiopentone and succinylcholine, desflurane was administered for maintenance of anaesthesia. Ten

Effects of hyperthermia and stimulation of the hypothalamus on the activity of the phrenic nerve in hypo- normo- and hypercapnic rabbits.

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Experiments were carried out on male rabbits, anesthetized with urethane, bilaterally vagotomized, paralysed and artificially ventilated. The preoptic area (POA) was stimulated at three different levels of CO2: in hypo- normo- and hypercapnia under normothermia, moderate hyperthermia and

Influence of body temperature on responses to hypoxia and hypercapnia: implications for SIDS.

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1. This paper reviews current knowledge regarding interactions between body temperature and the respiratory responses to hypoxia and/or hypercapnia, with special emphasis on how these interactions might predispose towards sudden infant death syndrome (SIDS). 2. Use has been made of an adult rat

Anemia, hypoxia and hypercapnia thresholds. Lessons from physiological limits in critically ill patients.

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Physiological alterations occur in the critical care medicine and reflect illness. Rendering patients physiologic parameters in the range that is normal for the population is not necessarily good; it may be frankly harmful. We do not currently possess outcome-based tools that allow us to titrate

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