keloid/glutathione

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Downregulation of apoptosis-related genes in keloid tissues.

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BACKGROUND Physiologically programmed cell death or apoptosis occurs during the natural balance between cellular proliferation and demise. METHODS We compared the expression of 64 apoptosis-related genes in keloids and normal scars to investigate the potential role of apoptosis in keloid formation.

A novel truncated TGF-beta receptor II downregulates collagen synthesis and TGF-beta I secretion of keloid fibroblasts.

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Hypertrophic scars and keloid are dermal proliferative disorders in wound healing. Transforming growth factor beta (TGF-beta) has been implicated in scar formation through the activation of fibroblasts and the acceleration of collagen deposition. Our study aimed to design a novel truncated (27-123

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