mitral valve prolapse/potassium

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[Anorexia/bulimia nervosa and mitral valve prolapse in a marathon runner].

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A 23-year-old male student (height 180 cm, weight 63 kg) without any cardiac symptoms underwent a cardiological examination and consultation before a marathon run. A holosystolic mitral valve prolapse was discovered. After the marathon run he developed anorexia nervosa, interrupted by brief bulimic

[Electrolyte abnormalities and ventricular arrhythmias in children with mitral valve prolapse].

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The aim of this study was to determine plasma concentration of magnesium, potassium, calcium and sodium in children with mitral valve prolapse (MVP) and to establish the relation between electrolyte abnormalities and ventricular arrhythmias (VA). The study group consists of the 113 children with MVP

[Influence of autonomic nervous system on electrolyte abnormalities in children with mitral valve prolapse].

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Measurement of the heart rate variability enables to assess the activity of the autonomous nervous system. The aim of the study was to evaluate the possible relationship between clinical status, serum sodium (Na), potassium (K), calcium (Ca), magnesium concentrations and HRV in children with mitral

Alarming atrioventricular block and mitral valve prolapse in the Kearns-Sayre syndrome.

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Kearns-Sayre syndrome (KSS) is a multisystem mitochondrial disorder characterized by the invariant triad: onset before 20, progressive external ophthalmoplegia and pigmentary retinal degeneration, plus at least one of the following: complete (or not) heart block, cereberal dysfunction and CSF

Normal left ventricular performance in children with X-linked hypophosphatemic rickets: a Doppler echocardiography study.

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To determine if chronic hypophosphatemia causes myocardial dysfunction, we explored one model for this metabolic derangement by prospectively investigating 11 patients (aged 5-18 years) with X-linked hypophosphatemic rickets (XLH) by M-mode, two-dimensional, and Doppler echocardiography. Inorganic

[Risk factors for sudden cardiac death related to the long QT syndrome].

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BACKGROUND Long QT syndrome is a rare condition. The syndrome is characterised by a prolonged QT-interval corrected for heart rate (QTc). The typical clinical presentation is the occurrence of syncope or cardiac arrest in young and otherwise healthy individuals. Cardiac events can be precipitated by

Upper limb ischemia induced by chronic licorice ingestion.

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A young woman had an acute embolic episode that affected her right upper limb. An axillo-brachial thromboembolectomy was performed after which distal pulses were recovered. The patient had a long lasting history, longer than ten years, of continuous licorice ingestion. Blood samples showed severe

Severe Mitral Regurgitation As a Result of Rupture of Mitral Valve Chordae Tendineae in a Patient With Graves Disease.

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Mitral valve prolapse is a common disorder, but severe mitral regurgitation (MR) as a result of rupture of mitral valve chordae tendineae is a rare manifestation of thyrotoxic heart disease. There are limited reports with respect to the onset of severe MR as a complication of Graves disease. We

[Magnesium deficiencies in cardiovascular diseases].

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Plasma (pl), red blood cell (ery) and urinary magnesium (Mg) concentrations were measured by spectrophotometry in over 300 patients at the Cardiology Department of Broussais Hospital. Other biological parameters, including pl and ery potassium, calcium and phosphate concentrations were measured

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