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anemia/phosphatase
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Chicken anemia virus.
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Chicken anemia virus (CAV), the only member of the genus Gyrovirus of the Circoviridae, is a ubiquitous pathogen of chickens and has a worldwide distribution. CAV shares some similarities with Torque teno virus (TTV) and Torque teno mini virus (TTMV) such as coding for a protein inducing apoptosis
[Autoimmune hemolytic anemia anemia followed by acute granulocytic leukemia (author's transl)].
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The authors report a case of acute myeloid leukemia developping 5 years after the onset of an autoimmune hemolytic anemia. The cytological origin of the proliferation is difficult to assess: the presence of prominent membranous complexes and a strong positivity of acid phosphatase reaction favour
Leukocyte alkaline phosphatase in sickle-cell anemia.
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Serum alkaline phosphatase isoenzymes in sickle cell anemia.
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Alkaline phosphatase isoenzymes in sera from patients with sickle cell anemia were separated by electrophoresis on starch gel. Physical and biochemical criteria identified bone alkaline phosphatase as the principal, although not necessarily the sole, enzyme fraction that increases during symptomatic
Bendamustine-associated hemolytic anemia.
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OBJECTIVE
To report a case of probable bendamustine-related hemolytic anemia.
METHODS
A 64-year-old white female had recently received treatment with bendamustine for stage III follicular lymphoma. After her fourth cycle, she was admitted to an outside facility with severe right upper quadrant pain
Anemia in primary hyperparathyroidism.
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Although anemia has not been widely appreciated as a complication of primary hyperparathyroidism, 5.1% of the individuals with this disorder seen at the Massachusetts General Hospital since 1962 had a normochromic, normocytic anemia that could not be related to blood loss,a deficiency state, or
Low leukocyte alkaline phosphatase activity in sickle cell anemia.
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Hemolytic Anemia: Sneaky Cause, Leaky Valve
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Intravascular hemolysis is a known complication of prosthetic heart valves. Severe hemolysis is rare (<1%) with the use of newer generation prosthetic valves. This usually occurs due to paravalvular leaks (PVLs). We present a case of hyperbilirubinemia and hemolytic anemia occurring as a result
[Studies of the acid phosphatase activity of erythrocytes in anemia].
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Letter: Decreased leukocyte alkaline phosphatase activity in sickle-cell anemia.
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Hyperparathyroidism and anemia in renal failure.
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Patients with severe secondary hyperparathyroidism, usually associated with osteitis fibrosa on bone histology, show considerable resistance to Epoetin, partly because of replacement of the cellular components of the bone marrow by fibrous tissue. In case of unexplained resistance to Epoetin,
Oxidative stress-associated protein tyrosine kinases and phosphatases in Fanconi anemia.
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CONCLUSIONS
Fanconi anemia (FA) is a genetic disorder featuring chromosomal instability, developmental defects, progressive bone marrow failure, and predisposition to cancer. Besides the predominant role in DNA damage response and/or repair, many studies have linked FA proteins to oxidative stress.
Chicken anemia virus VP2 is a novel dual specificity protein phosphatase.
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The function of viral protein 2 (VP2) of the immunosuppressive circovirus chicken anemia virus (CAV) has not yet been established. We show that the CAV VP2 amino acid sequence has some similarity to a number of eukaryotic, receptor, protein-tyrosine phosphatase (PTPase) alpha proteins as well as to
Chronic kidney disease-associated anemia: new remedies.
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Erythropoiesis stimulating agents (ESAs) are effective drugs that correct anemia in patients with chronic kidney disease (CKD). Recombinant human erythropoietin (EPO), the first ESA that became available more than 20 years ago, is similar to the naturally occurring molecule. In subsequent years,
Abnormal red cell membrane phosphatase activity in an unidentified congenital hemolytic anemia.
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The erythrocyte membrane contains a neutral phosphatase, which was studied with p-nitrophenyl-phosphate as the substrate. The enzyme was investigated in four members of the same family, suffering from a congenital spherocytic hemolytic anemia. Although the condition was transmitted as a dominant
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