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glyoxylic acid/atrofia
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Altered histofluorescent pattern of noradrenergic innervation of the cerebellum of the mutant mouse Purkinje cell degeneration.
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The Purkinje target cells for noradrenergic fibers originating in the locus coeruleus are considered to be of importance in the regulation of noradrenergic input to the cerebellum. The availability of a mouse mutant, Purkinje cell degeneration provides a non-surgical means for studying cellular
Wallerian degeneration is required for both neuropathic pain and sympathetic sprouting into the DRG.
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Chronic loose constriction of the sciatic nerve produces mechanoallodynia and thermal hyperalgesia in rats and mice, and the behaviour develops during the time in which the nerve distal to the ligature site is undergoing Wallerian degeneration. There is a sympathetic component to the pain generated
Noradrenergic innervation of the cerebellar cortex in normal and in Purkinje cell degeneration mutant mice: evidence for long term survival following loss of the two major cerebellar cortical neuronal populations.
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Purkinje cell degeneration mutant mice were examined during the course of Purkinje cell death (26 and 35 days old) and at 3, 5, 9 and 12 months of age. Glyoxylic acid fluorescence histochemistry for catecholamines was used to investigate possible alterations or reorganization of the noradrenergic
Evidence for exclusive adrenergic innervation of feather muscles (mm. pennati) in the chicken. Histochemical studies and experiments with 5-hydroxydopamine.
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Feather follicles in the avian skin are interconnected by well-defined bundles of smooth muscle cells, which are responsible for the erection and depression of feathers and thus play an important role in thermoregulation. The depressing and erecting muscle bundles were found to receive a very dense
Effects of streptozotocin diabetes on the noradrenergic innervation of the rat heart: a longitudinal histofluorescence and neurochemical study.
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The effects of the age of induction and total duration of streptozotocin diabetes on the sympathetic noradrenergic innervation of the rat heart was examined with glyoxylic acid induced histofluorescence to demonstrate the distribution of noradrenergic fibers within the heart, and with high
Regenerative catecholamine-containing terminals in kitten visual cortex: an ultrastructural study.
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Ultrastructure of catecholaminergic (CA) terminals was studied in the kitten visual cortex which had been continuously infused with 6-hydroxydopamine (6-OHDA) for one week. Two methods were used to identify CA terminals: (1) cardiac perfusion with glyoxylic acid (GA) followed by potassium
Noradrenergic sprouting and beta-adrenergic receptor binding in the lateral geniculate nucleus of rats after unilateral visual cortex ablation.
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One and two weeks after unilateral visual cortex (VC) ablation beta-adrenergic receptor binding is increased in the lateral geniculate nucleus (LGN) of both sides. 6 month later beta-receptor binding in the LGN is decreased again and no differences to untreated control animals are detectable. Using
Fluorescence histochemical and electron-microscopical observations on the innervation of the atrial myocardium of the adult human heart.
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The existence of both adrenergic and cholinergic innervation of the atrial myocardium of the adult human heart was demonstrated by means of fluorescence induced by formaldehyde or glyoxylic acid and by electron microscopy. The adrenergic fluorescing axons (1) followed the course of blood vessels as
In vivo and in vitro studies on the neurotoxic potential of 6-hydroxydopamine analogs.
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In an attempt to determine which physical and biological properties could best be correlated with neurotoxic potential, seven analogs of 1-(2,4,5-trihydroxyphenyl)-2-aminoethane (1), better known as 6-hydroxydopamine, were synthesized and compared to 1 in a variety of ways both in vivo and in vitro.
Striatal afferents in the newt Triturus cristatus.
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In order to investigate the neuronal populations projecting to the corpus striatum in the brain of a urodele, Triturus cristatus, horseradish peroxidase (HRP) retrograde labeling was used in parallel with anterograde degeneration, glyoxylic acid histofluorescence and behavioral testing. Striatal
The nerve response after autotransplantation of the rabbit ear.
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Neurohistologic investigations on 16 autotransplants of the rabbit ear ranging from 3 weeks to 18 months after replantation are described. This study investigated degeneration and regeneration of the adrenergic nerves of the vascular system and the afferent endings in hairy skin. Histochemical
A quantitative study of rat uterine sympathetic innervation during pregnancy and post partum.
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In mammals, pregnancy induces a transient and extensive degeneration of uterine sympathetic innervation. We used the models of unilateral oviduct ligation and in oculo myometrium transplant in pregnant rats to address the role of stretching forces and/or hormone milieu in the loss of sympathetic
The regeneration of the sympathetic catecholaminergic nerve fibers in the process of burn wound healing in guinea pigs.
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In the present study, morphological alterations of cutaneous catecholaminergic nerve fibers in the process of burn wound healing have been investigated by using glyoxylic acid induced fluorescence histochemistry. The sympathetic denervation is detected within 48 hr after burns as well as in various
Intracranial self-stimulation from the sulcal prefrontal cortex in the rat: the effect of 6-hydroxydopamine or kainic acid lesions at the site of stimulation.
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An electrode cannula system was used to elicit intracranial self-stimulation (ICSS) from the sulcal prefrontal cortex in rats to test the behavioral effects of local infusions of 6-hydroxydopamine (6-OHDA) or kainic acid (KA) into the brain area surrounding the electrode tip. In experiment I sulcal
Extracellular matrix molecules influence innervation density in rat cerebral blood vessels.
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We have investigated the contribution of the acellular component (i.e., the extracellular matrix, or ECM) in determining the innervation density of peripheral target tissue, and the possible involvement of ECM molecules in age-related axonal atrophy. We addressed this issue using cerebral blood
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