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odontogenic tumors/phosphatase
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12 resultados
Ultrastructural localization of alkaline phosphatase in the calcifying epithelial odontogenic tumor.
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The localization of alkaline phosphatase in a calcifying epithelial odontogenic tumor obtained from a 53-year-old man was examined cytochemically. The majority of enzyme activity was associated with the epithelial cell membranes of the tumor, and faint activity was found in the cell membranes facing
[Histochemical and immunohistochemical double staining for osteolytic related factors in keratocystic odontogenic tumor].
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OBJECTIVE
To investigate the activities of bone resorption related cytokines at the capsule wall of the keratocystic odontogenic tumor (KCOT) by histochemical and immunohistochemical double staining for tartrate-resistant acid phosphatase staining (TRAP), receptor activator of nuclear factor-kappaB
Clear cell odontogenic tumor: histochemical and ultrastructural features.
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Clear cell odontogenic tumor, a rare epithelial jaw lesion of putative odontogenic origin, histologically resembles clear cell adenocarcinomas. Ultrastructural and histochemical features are described and support a non-glandular derivation. The intraosseous neoplasm is characterized by ovoid nests
Histoenzymological and ultrastructural study of a bifocal calcifying epithelial odontogenic tumor. Characteristics of epithelial cells and histogenesis of amyloid-like material.
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A calcifying epithelial odontogenic tumor, simultaneously located in the two jaws (maxilla and mandible) was examined by histochemical and electron microscopic methods. Squamous tumor cells without secretory polarity were different from those of common ameloblastoma. High activities of alkaline
Fibroblasts regulate variable aggressiveness of syndromic keratocystic and non-syndromic odontogenic tumors.
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Keratocystic odontogenic tumors (KCOTs) are jaw lesions that can be either sporadic or associated with nevoid basal cell carcinoma syndrome, which typically occurs as multiple, aggressive lesions that can lead to large areas of bone destruction and resorption and cause major impairment and even jaw
The effects of marsupialization on bone regeneration adjacent to keratocystic odontogenic tumors, and the mechanisms involved.
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Bone resorption in the jaws is one of the most severe complications of keratocystic odontogenic tumors (KCOTs), and can be treated by either enucleation or marsupialization. However, the effects of marsupialization on bone regeneration adjacent to KCOTs, and the mechanisms involved, are still
Ameloblastic fibrosarcoma of the jaws--report of three cases. Clinico-pathologic, histoenzymological and ultrastructural study.
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The ameloblastic fibrosarcoma is a rare variety of neoplasm. Three new cases reported here occurred within preexistent benign odontogenic tumors (ameloblastic fibroma or fibro-odontoma). These large, osteolytic tumors, spreading to adjacent soft parts, recurred after surgical treatment in two cases.
Surface vacuolar ATPase in ameloblastoma contributes to tumor invasion of the jaw bone.
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Ameloblastoma is the most common benign odontogenic tumor in Japan. It is believed that it expands in the jaw bone through peritumoral activation of osteoclasts by receptor activator of nuclear factor kappa-B ligand (RANKL) released from the ameloblastoma, as in bone metastases of cancer cells.
[Odontogenic induction and ameloblastoma. Histoenzymological and ultrastructural studies (author's transl)].
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The ameloblastoma is usually thought to be devoid of any odontogenic capacity. However, the histological, ultrastructural and histoenzymological study of 5 cases demonstrates the fairly high level of differentiation of some ameloblastic cells in such neoplasms. Furthermore, it suggests very early
Establishment of mesenchymal cell line derived from human developing odontoma.
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OBJECTIVE
An odontoma, which shows proliferating odontogenic epithelium and mesenchymal tissue, is one of the most common odontogenic tumors encountered. These are commonly found in tooth-bearing regions, although the etiology remains unknown. There are no previous reports of an established line of
[Odontogenic myxoma. A histoenzymologic and ultrastructural study. Apropos of 5 cases].
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The myxoma of the jaws represent 8% of the odontogenic tumours in our series (324 cases). The 5 cases described here were studied by histoenzymological and electron microscopic methods. The first method demonstrated in the majority of tumoral cells a high level of activity of alkaline phosphatases
Immortalization of human dental papilla, dental pulp, periodontal ligament cells and gingival fibroblasts by telomerase reverse transcriptase.
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BACKGROUND
Human telomerase reverse transcriptase (hTERT) is catalytic subunit of human telomerase.
METHODS
We studied the immortalization of a series of human dental and periodontal cells by ectopic expression of hTERT and co-expression of hTERT with human papilloma virus 16 (HPV16) or simian virus
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