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phosphatidyl ethanolamine/hypoxia
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6 resultados
Changes in phospholipids and free fatty acids in the brains of mice preconditioned by hypoxia.
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Changes in the composition and contents of phospholipids and free fatty acids were observed and compared in three groups: (A) unpreconditoned normal controls, (B) exposure to 1 run of hypoxia and (C) exposure to 4 runs of hypoxia. In group B, the content of phosphatidyl ethanolamine (PE),
Effect of acute hypobaric hypoxia on 32-P incorporation into phospholipids of alveolar surfactant, lung, liver and plasma of rat.
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Exposure of adult rats to hypobaric hypoxia caused hypolipidemia, hypotriglyceridemia and hypophospholipidemia. Hypobaric hypoxia produced an increase in liver triglyceride and cholesterol levels and a decrease in lung triglyceride, total phospholipid and phosphatidyl choline. The proportion of
[Changes in mitochondrial lipid composition in hypoxia in the presence of a dinitrophenol uncoupler].
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Incubation of mitochondria under hypoxic conditions in hypotonic solution of sucrose containing dinitrophenol as an uncoupler of oxidative phosphorylation led to hydrolysis of mitochondrial phospholipids, with the most pronounced destruction of phosphatidyl ethanolamine and cardiolipin fractions.
Determination of membrane lipid differences in insulin resistant diabetes mellitus type 2 in whites and blacks.
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OBJECTIVE
Insulin resistance in diabetes mellitus type 2 (DM2) can result from membrane lipid alterations. Blacks are at a higher risk of developing DM2; therefore, we investigated whether membrane lipid differences exist between blacks and whites and if differences contribute to impaired insulin
Interaction of phosphatidylserine-phosphatidylcholine liposomes with sickle erythrocytes. Evidence for altered membrane surface properties.
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The sickle erythrocyte (RBC) is a pathologic RBC that contains multiple membrane abnormalities. Some of these abnormalities have been implicated in the pathophysiology of vasoocclusive crises characteristic of sickle cell disease; others have yet to be defined in terms of their clinical
Alveolar stability of altitude raised rats.
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On the lining of alveolar membrane of the lung presence of surfactant, a surface tension lowering agent which is a phospholipid in nature, is well established. A good correlation exists between pulmonary lecithin, the principle constituent of surfactant system and alveolar stability. The production
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