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phosphatidyl serine/hemorragia
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Nonsurgical bleeding diathesis in anemic thrombocytopenic patients: role of temperature, red blood cells, platelets, and plasma-clotting proteins.
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Research at the Naval Blood Research Laboratory (Boston, MA) for the past four decades has focused on the preservation of red blood cells (RBCs), platelets (PLTs), and plasma-clotting proteins to treat wounded servicemen suffering blood loss. We have studied the survival and function of fresh and
[Erythrocyte lipids and the gas transport function of blood in acute hemorrhage].
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Alterations of erythrocyte lipid composition were studied during oxygenation of the cells in rabbit lesser circulation in simultaneous single massive hemorrhage. The state of blood respiratory function was studied in order to evaluate an importance of the erythrocyte membranes transformation
Use of flow cytometry in an apoptosis assay to determine pH and temperature stability of shiga-like toxin 1.
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Shiga toxins and Shiga-like toxins (Stx) are a relatively large group of cytotoxins produced by certain serotypes of Shigella and E. coli (STEC). These toxins are responsible for diarrhea, hemorrhagic colitis and may induce hemolytic uremic syndrome (HUS) with serious consequences in young children.
Massive transfusion: an overview of the main characteristics and potential risks associated with substances used for correction of a coagulopathy.
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Massive transfusion (MT) is an empiric mode of treatment advocated for uncontrolled bleeding and massive haemorrhage, aiming at optimal resuscitation and aggressive correction of coagulopathy. Conventional guidelines recommend early administration of crystalloids and colloids in conjunction with red
Platelet CD36 mediates interactions with endothelial cell-derived microparticles and contributes to thrombosis in mice.
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CD36 is a scavenger receptor that binds multiple ligands, including phosphatidyl serine (PS). Although CD36(-) mice do not have a bleeding diathesis, we show here that they do have significantly prolonged thrombotic occlusion times in response to FeCl(3)-induced vascular injury. Because cell-derived
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