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Studies on necrosis of mouse liver in vitro. Alterations in activities of succinoxidase, succinic dehydrogenase, glutamic dehydrogenase, acid phosphatase, uricase, glucose-6-phosphatase and NAD-pyrophosphorylase.
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THE RELATION OF FATTY DEGENERATION TO THE OXIDATION OF PURINES BY LIVER CELLS.
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These experiments show that it is possible to cause, by the proper use of hydrazine and phosphorus, a very high degree of fatty change in the liver with a minimum of necrosis, involving alike both peripheral and central portions of the lobule and thus eliminating the " factor of safety" or residual
Adenosine A2A receptors and uric acid mediate protective effects of inosine against TNBS-induced colitis in rats.
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Inflammatory bowel disease comprises chronic recurrent inflammation of gastrointestinal tract. This study was conducted to investigate inosine, a potent immunomodulator, in 2,4,6-trinitrobenzene sulphonic acid (TNBS)-induced chronic model of experimental colitis, and contribution of adenosine A(2A)
Is there association between uric acid and inflammation in hemodialysis patients?
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BACKGROUND
Elevated serum uric acid has been associated with a variety of cardiovascular disease and with inflammation, but these have been little explored in chronic kidney disease (CKD). Elevated uric acid levels are common in CKD patients and could be involved in inflammatory milieu; our aim was
Elevated serum uric acid is associated with high circulating inflammatory cytokines in the population-based Colaus study.
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BACKGROUND
The relation of serum uric acid (SUA) with systemic inflammation has been little explored in humans and results have been inconsistent. We analyzed the association between SUA and circulating levels of interleukin-6 (IL-6), interleukin-1β (IL-1β), tumor necrosis factor- α (TNF-α) and
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