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allyl alcohol/hypoxia

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ArtiklidKliinilistes uuringutesPatendid
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Exposure of rats to a reduced oxygen tension (6% O2, 94% N2) for 6 h increased the serum enzyme and the histological lesions induced by carbon tetrachloride (CCl4). Hypoxia did not enhance the hepatotoxic response to paracetamol, allyl alcohol, bromobenzene or thioacetamide. No correlation was found
Kupffer cells play an important role in liver function and phagocytosis of foreign particles in the hepatic portal tract. Therefore, the purpose of this study was to investigate the influence of several hepatotoxic chemicals (allyl alcohol, ethylhexanol, and menadione) and hypoxia on phagocytic
Spores of Bacillus megaterium were irradiated in suspension with 50 kVp X-rays under three reference conditions: in anoxia (i.e., 100 per cent N2); in anoxia with 2mM p-nitroacetophenone (PNAP), a concentration that shows the maximum amount of sensitization by this agent; and in air. The responses

Influence of 2,4-dinitrophenol on the susceptibility of rats to hepatotoxic injury.

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Treatment of rats with 2,4-dinitrophenol (DNP) markedly enhanced the release of enzymes into serum induced by carbon tetrachloride (CCl4). DNP also aggravated the hepatotoxic response to paracetamol but not that to allyl alcohol, bromobenzene and thioacetamide. DNP-induced hypoxia resulting in an
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