chrysanthemum chanetii/hypoxia

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Chrysanthemum morifolium extract improves hypertension-induced cardiac hypertrophy in rats by reduction of blood pressure and inhibition of myocardial hypoxia inducible factor-1alpha expression.

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BACKGROUND Chrysanthemum morifolium Ramat. (Asteraceae) extract (CME) possesses a vasodilator effect in vitro. However, the use of polyphenol-rich CME in the treatment of hypertension-induced cardiac hypertrophy has not been reported. OBJECTIVE We investigated the effect of polyphenol-rich CME on

Chrysanthemum morifolium attenuated the reduction of contraction of isolated rat heart and cardiomyocytes induced by ischemia/reperfusion.

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The present study was aimed to investigate the effect of Chrysanthemum morifolium Ramat. (CM) on isolated rat heart and ventricular myocytes during ischemia/anoxia and reperfusion/reoxygenation. The ischemia/reperfusion injury was induced by ligation the left artery descending coronary of isolated

Antioxidant activities of aqueous extracts from 12 Chinese edible flowers in vitro and in vivo.

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The antioxidant function of edible flowers have attracted increasing interest. However, information is lacking on the impact of edible flowers on oxidative injury including hypoxia-re-oxygenation and hyperlipidemia. The antioxidant activities of aqueous extracts from 12 Chinese edible flowers were

Over-expression of CmSOS1 confers waterlogging tolerance in Chrysanthemum.

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Manipulating the level of expression of SOS1, a protein which regulates the movement and distribution of sodium ions, has been shown to enhance the salinity tolerance of a number of plant species, but its involvement in the response to hypoxia is less well established. Here, the contribution of the

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