hypoparathyroidism/seizures

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Recurrent seizures, mental retardation and extensive brain calcinosis related to delayed diagnosis of hypoparathyroidism in an adolescent boy.

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Reports of adolescent patients presenting with intractable seizures and mental retardation secondary to idiopathic hypothyroidism are uncommon in the literature. In this case, we report a 17-year-old boy who developed recurrent seizures, mental retardation and extensive brain calcinosis related to

A Case of Seizure Revealing Fahr's Syndrome with Primary Hypoparathyroidism.

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BACKGROUND Idiopathic basal ganglia calcification, also known as Fahr's disease or Fahr's syndrome, is a rare neurological disorder characterized by abnormal calcified deposits in the basal ganglia. Here, we report a case of Fahr's syndrome with calcification of the basal ganglia due to

Seizures and Basal Ganglia Calcification Caused by Hypoparathyroidism

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Seizures, not a typical feature of hypoparathyroidism, may present along with myalgia and tetany in patients of hypoparathyroidism. Thorough history and examination, derangement of biochemical parameters such as hypocalcemia, hyperphosphatemia, and inappropriately low levels of parathyroid hormone,

Hypoparathyroidism: an unusual cause of seizures.

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A 73-year-old woman with hypoparathyroidism and hypocalcemic seizures was treated successfully with intravenous calcium. These symptoms developed 19 years after thyroid surgery. Prior to this admission, there had been many opportunities to make the diagnosis, had it been considered. Chronic

Congenital hypoparathyroidism presenting as recurrent seizures in an adult.

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Hypocalcemia due to hypoparathyroidism may manifest as serious neurologic symptoms such as seizures, movement disorders, or raised intracranial pressure. Several patients were observed to have these dangerous neurologic complications even without subtle signs of hypocalcemia like tetany, chvostek's

[Hypocalcemia and neonatal seizures: a rare case of congenital hypoparathyroidism].

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We report the case of a white male infant, 2 months-old, with tremor and hypertonia since 15th day of life transferred to our service and diagnosed as seizures. Investigation showed hypocalcaemia (4 mg/dl) and hypomagnesemia (1.6 mg/dL) and the infant's serum metabolic disturb was corrected by

Congenital hypoparathyroidism, seizure, extreme growth failure with developmental delay and dysmorphic features--another case of this new syndrome.

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A 4-year-old Saudi female child with extreme failure to thrive, striking dysmorphic features, developmental delay, congenital hypoparathyroidism, UTI, seizures, chronic otitis media, chronic non-specific gastroenteritis and repeated life-threatening infections was followed from birth. She was the

Infantile convulsions and paroxysmal kinesigenic choreoathetosis in a patient with idiopathic hypoparathyroidism.

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We reported a 15-year-old boy with idiopathic hypoparathyroidism who presented with paroxysmal kinesigenic choreoathetosis at age 10. Calcium levels were low and intact parathyroid hormones were undetectable in serum. Computed tomography showed calcifications in the basal ganglia, thalamus, and

[Epileptic seizure as the first sign of hypoparathyroidism].

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BACKGROUND Hypoparathyroidism refers to a group of disorders in which extracellular calcium levels cannot be maintained within the normal range due to relative or absolute deficiency of parathyroid hormone (PTH). The clinical features of hypoparathyroidism are consistent with hypocalcaemia and,

Epileptic seizure, as the first symptom of hypoparathyroidism in children, does not require antiepileptic drugs.

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OBJECTIVE Patients with hypoparathyroidism exhibit metabolic disorders (hypocalcemia) and brain structural abnormalities (brain calcifications). Currently, studies have determined whether antiepileptic drug (AED) treatment is required for epileptic seizures in children with

Hypoparathyroidism Causing Seizures: When Epilepsy Does Not Fit.

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A 24-year-old man presented to the Chris Hani Baragwanath Academic Hospital emergency department with recurrent seizures having previously been diagnosed with epilepsy from age 14. The biochemical investigations and brain imaging were suggestive of seizures secondary to hypocalcemia, and a diagnosis

[Idiopathic hypoparathyroidism and convulsive seizures].

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In a patient with primary hypoparathyroidism the disease ran for years only generalized convulsions resistant to antiepileptic medication, and was lately complicated by bilateral cataracts. Low serum calcium and elevated serum phosphorus found on occasion of a grand mal status resistant to routine

Neonatal idiopathic primary hypoparathyroidism: A rare cause of neonatal seizures.

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Etiology of neonatal seizures (NNS) is diverse and hypocalcemia is one of the treatable causes. Neonatal hypocalcemia (NHC) due to congenital hypoparathyroidism, either permanent or transient, is extremely rare. Its biochemical abnormalities include hypocalcemia, hyperphosphatemia and low levels of

Seizures in patients with idiopathic hypoparathyroidism: effect of antiepileptic drug withdrawal on recurrence of seizures and serum calcium control.

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OBJECTIVE There is limited information on seizures in patients with idiopathic hypoparathyroidism (IH). We assessed seizure characteristics at presentation, subclinical seizures during follow-up, and the effect of antiepileptic drug (AED) withdrawal in IH patients. METHODS Seizure characteristics

Proton pump inhibitor-induced hypocalcemic seizure in a patient with hypoparathyroidism.

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OBJECTIVE To report a case of proton pump inhibitor-induced hypocalcemic seizure in a patient with hypoparathyroidism. METHODS We describe the clinical history, physical examination findings, and laboratory values of the patient and briefly review the relevant literature. RESULTS A 48-year-old woman

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