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oxalate/väsimus

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Wu-Ling-San (WLS) formula has been proved to prevent calcium oxalate nephrolithiasis both in vitro and in vivo. This is the first prospective, randomized and placebo-controlled clinical trial of WLS in calcium oxalate nephrolithiasis prevention. All patients who enrolled were asked to drink enough

Acute oxalate nephropathy due to high vitamin C doses and exocrine pancreatic insufficiency.

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Oxalate kidney injury can manifest as oxalate nephropathy or nephrolithiasis and present as acute kidney injury or even as end-stage renal disease. There are several known causes for acute oxalate nephropathy; however, the combination of exocrine pancreatic insufficiency with overconsumption of

Coating of enamel and dentin with bioactive materials: Effect on adhesive interface of class V restorations.

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To evaluate the effect of coating enamel and dentin with a calcium phosphate powder, a calcium fluoride silicon polymer, an oxalate and a functionalized tricalcium phosphate, on marginal adaptation of class V cavities restored with a universal adhesive containing the adhesive monomer
Inborn errors of metabolism cause increase of metabolites in serum and their deposition in various organs including bone marrow. Primary hyperoxaluria (PH) is a rare inborn error in the pathway of glyoxylate metabolism which causes excessive oxalate production. The disease is characterized by
BACKGROUND Comorbid depression and anxiety may result in greater symptom severity and poorer treatment response than either condition alone. Selective serotonin reuptake inhibitors have been found to be effective in treating both depression and anxiety; however, pharmacodynamic and pharmacokinetic

A method for bonding to dentin and enamel.

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In vitro tensile bond strengths of about 1 ton per square inch between composites and dentin and enamel were obtained by applying a 5.3% solution of ferric oxalate to the tooth surface, followed by water and air cleaning; by applying a 10% solution of NTG-GMA, followed by cleansing with solvent and
Primary hyperoxaluria (PH) is a rare genetic disorder characterized by overproduction of oxalate due to specific enzyme deficiencies in glyoxylate metabolism. The primary clinical presentation is in the form of recurrent urolithiasis, progressive nephrocalcinosis, end-stage renal disease, and

Sarcoidosis presenting as granulomatous myositis in a 16-year-old adolescent.

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BACKGROUND Sarcoidosis is a multi-system disease characterized by the presence of non-caseating epithelioid granulomas in affected tissues, including skeletal muscle. These organized collections of immune cells have important pathophysiologic action including cytokine production leading to
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