thiamin/hypoxia

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Lactic Acidosis Secondary to Thiamin Deficiency Following Autologous Stem Cell Transplantation

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A 22-month-old female with high-risk neuroblastoma completed 5 cycles of chemotherapy then underwent high-dose chemotherapy with autologous stem cell rescue (ASCR). Parenteral nutrition was administered from day +2 following ASCR, as she was unable to tolerate nasogastric feeds because of grade IV

Thiamin absorption by small intestine.

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In different animal species, including man, thiamin is absorbed in vivo from the small intestine by means of two mechanism: at low intraluminal concentrations (less than 1.5 microM), absorption shows a saturation kinetics; at higher concentrations, passive transport (diffusion) takes place. The

[Characteristics of thiamine diphosphate metabolism in the liver in acute hypobaric hypoxia].

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Rates of thiamin diphosphate accumulation in rat liver tissue and its subsequent depletion were decreased within the first hour after vitamin B1 administration into the animals with oxygen insufficiency. Hypoxia apparently inhibited simultaneously the processes of thiamin diphosphate biosynthesis

Sudden infant death syndrome and abnormal metabolism of thiamin.

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Although it has been generally accepted that moving the infant from the prone to the supine position has solved the problem of sudden infant death syndrome (SIDS), it has been hypothesized that this is an insufficient explanation and that a mixture of genetic risk, some form of stressful incident

[The transketolase activity in erythrocytes as a criterion for thiamine balance in the body].

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In deficiency of thiamin the transketolase activity was decreased in patients and in experimental animals. The data obtained showed the high sensitivity of transketolase from erythrocytes to hypovitaminosis B1. At the same time, in diseases, especially accompained by hypoxia (and also by

[Stereometric evaluation of the myocardial cardiomyocyte-capillary ratio in emotional and pain-induced stress after administration of thiamine and nicotinamide].

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The effects of thiamin and nicotinamide on left-ventricular myocardial cardiomyocyte/capillary ratio at the outcome of lasting emotional/painful stress have been examined in rats, using stereometric techniques. Thiamin is shown to be capable of preventing, to a certain extent, myocardial

Dopamine, acetylcholine, and glutamate interactions in aging. Behavioral and neurochemical correlates.

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Aging, hypoxia, and thiamin deficiency diminish motor performance. Similar alterations of ACh, DA, and glutamate metabolism accompany hypoxia, thiamin deficiency, and aging. Both aging and hypoxia reduce ACh release and stimulate DA and glutamate release. Presynaptic enhancement of DA and glutamate

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Herbal & Natural Medicine

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