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Sivu 1 alkaen 23 tuloksia
Bilateral striopallidodentate calcification (Fahr's syndrome) and multiple system atrophy in a patient with longstanding hypoparathyroidism.
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Recently, a family with idiopathic brain calcification was reported, in which one family member was diagnosed with multiple system atrophy (MSA) at autopsy. We report here a case showing similar neuropathological features in a patient with longstanding hypoparathyroidism. Our female patient had a
[Tetany as a sole manifestation in a patient with Bartter's syndrome and a successful treatment with indomethacin].
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37 year-old man presented to our clinic because of tetany that occurred during driving a car in the morning. He had no anorexia, chronic diarrhea or vomiting. He was normotensive and no edema was noted. On neurological examination, he was rather apprehensive and Trousseau sign was mildly positive.
The developmental neurotoxicity of fipronil: notochord degeneration and locomotor defects in zebrafish embryos and larvae.
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Fipronil is a phenylpyrazole insecticide designed to selectively inhibit insect gamma-aminobutyric acid (GABA) receptors. Although fipronil is often used in or near aquatic environments, few studies have assessed the effects of this neurotoxicant on aquatic vertebrates at sensitive life stages. We
Hypocalcemia and motor neuron degeneration in paraphysectomized frogs.
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The paraphysis has been studied extensively but its function is unknown. Surgical removal of the paraphysis in the bullfrog (Rana catesbeiana) resulted in hypocalcemia and tetany in 35 frogs, about 40% of those operated upon. One or two days after the onset of tetany (about 8 days postoperative),
Hypocalcemic myopathy without tetany due to idiopathic hypoparathyroidism: case report.
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Myopathy due to idiopathic hypoparathyroidism is very unusual. We report on a 30 years-old man referred with complaints of sporadic muscle pain and mild global weakness for 10 years. His physical examination showed normal strength in distal muscle and slightly weakness in the pelvic and scapular
Hyperexcitability of motor and sensory neurons in neuromyotonia.
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Two members of a family with a neuropathy resembling Charcot-Marie-Tooth disease were unable to relax their muscles after voluntary contraction. Muscle spasm often outlasted voluntary contraction by 30 seconds or more before subsiding into myokymia and fasciculations. The posture of the hand during
Nature of muscular change in osteomalacia: light- and electron-microscope observations.
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Thirteen muscle biopsy specimens (mainly the gluteus maximus) from 12 patients with laboratory confirmation of osteomalacia and proximal muscle weakness in 10 were examined by light and electron microscopy. Light microscopy revealed mild diffuse non-specific atrophy of the muscle fibres in 10 cases,
[Effect of parathyroidectomy on enamel formation of incisors in rats].
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A chronological study on the enamel formation of the incisors in male Wistar rats was undertaken following parathyroidectomy by using glyoxalbis (2-hydroxyanil) stain, ground sections, and scanning and transmission electron micrographs. In addition, the effect of activated vitamin D3 administration
THE RELATION BETWEEN THE THYROID AND PARATHYROID GLANDS.
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The following conclusions may be drawn from the experiments presented in this article. 1. Excessive meat diet develops hypertrophy of the thyroid gland. A definite hypertrophy of the parathyroid gland under the same conditions has not been established. A meat diet does not develop hypertrophy of the
Simazine toxicosis in sheep.
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A case of simazine toxicosis in sheep was investigated. Affected animals exhibited generalized muscle tremors which progressed to mild tetany followed by collapse of the hind legs. Other signs included a short prancing gait with head tucked in a similar manner to that of a show pony. Death occurred
THE INFLUENCE UPON THE SPLEEN AND THE THYROID OF THE COMPLETE REMOVAL OF THE EXTERNAL FUNCTION OF THE PANCREAS.
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The complete removal of the function of the pancreas concerned in digestion is followed by marked changes in the spleen and in the thyroid apparatus. Second, the spleen shows an extreme simple atrophy. Third, the thyroid apparatus exhibits a constant change shown by the macroscopic transparency of
Fasciculations in human hereditary disease.
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Fasciculations are a manifestation of peripheral nerve hyperexcitability in addition to myokymia, neuromyotonia, cramps, or tetany. Fasciculations occur in hereditary and non-hereditary diseases. Among the hereditary diseases, fasciculations are most frequently reported in familial amyotrophic
Myokymia and neuromyotonia in veterinary medicine: a comparison with peripheral nerve hyperexcitability syndrome in humans.
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Involuntary muscle hyperactivity can result from muscle or peripheral nerve hyperexcitability or central nervous system dysfunction. In humans, diseases causing hyperexcitability of peripheral nerves are grouped together under the term 'peripheral nerve hyperexcitability' (PNH). Hyperexcitability of
[Clinical analysis of 6 cases of Bartter syndrome].
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OBJECTIVE
To summarize the clinical characteristics of Bartter syndrome and investigate its pathogenesis.
METHODS
The clinical data of 6 cases of Bartter syndrome at our hospital from November 2006 to May 2010 were analyzed retrospectively.
RESULTS
The onset age of Bartter syndrome was 13-35 years
Fluorotoxic metabolic bone disease: an osteo-renal syndrome caused by excess fluoride ingestion in the tropics.
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BACKGROUND
There is scant data available on the pathogenetic mechanisms of varied clinical presentation of bone disease in patients with excess fluoride ingestion in the Indian subcontinent. The present study is comprehensive and state of the art, incorporating all essential elements of bone mineral
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