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Effects of ajmaline on right precordial Q waves simulating myocardial infarction.
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Seven patients with right precordial Q waves (RPQs) simulating anterior myocardial infarction (MI), but without a previous history of either MI or other underlying diseases showing RPQ, were injected intravenously with ajmaline. The RPQs were abolished in 5 patients. Two patients displayed a
[Comparative effects of ajmaline and lidocaine on hemodynamics in myocardial infarct].
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Haemodynamic changes after single intravenous injection of antiarhythmic doses of ajmaline (50 mg) and lidocain (100 mg) were measured comparatively in 10 patients with acute myocardial infarction, stable cardiac rhythm and without manifest left heart failure. The effects of ajmaline were
The efficacy of Ajmaline in ventricular arrhythmias after failure of lidocaine therapy in the acute phase of myocardial infarction.
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Forty-three patients in the acute phase of myocardial infarction who were resistant to conventional doses of lidocaine received Ajmaline intravenously (50 mg bolus followed by constant infusion rate of 1-1.5 mg/min). Dangerous ventricular arrhythmias were abolished in 72% of this group of patients
Effect of ajmaline on sustained ventricular tachycardia induced by programmed electrical stimulation in conscious dogs after myocardial infarction.
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As yet the antiarrhythmic efficacy of ajmaline with regard to suppressing the induction of sustained ventricular tachycardia after myocardial infarction has not been determined. Therefore, programmed electrical stimulation was performed in 8 conscious, chronically instrumented mongrel dogs 8-20 days
[Hemodynamic effects of xylocaine and ajmaline in myocardial infarction].
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[Hemodynamic effect of mesocaine and ajmaline in acute myocardial infarct].
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[ON AJMALINE TREATMENT OF PAROXYSMAL VENTRICULAR TACHYCARDIA IN MYOCARDIAL INFARCT].
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[Myocardial infarction and Wolff-Parkinson-White syndrome. Usefulness of the ajmaline test].
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[The action of ajmaline bitartrate on ventricular extrasystoles after recent myocardial infarction (author's transl)].
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[The prevention of arrhythmias in acute myocardial infarct. Use of ajmaline in continuous phleboclysis and of oral disopyramide].
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Ajmaline in non-responders to lidocaine with malignant ventricular arrhythmia after thrombolytic therapy in acute myocardial infarction.
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New aspects of the clinical use of anti-arrhythmia agents with special reference to acute therapy of ventricular tachycardia (lidocaine vs. ajmaline).
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Antiarrhythmic treatment is based on the hypothesis that ventricular premature beats (VPBs), in the presence of underlying cardiac disease and impaired ventricular function, may predispose to sudden cardiac death. The effectiveness of treatment, however, has not been proven. For acute treatment of
[Predictive value of the ajmaline test for the diagnosis of distal paroxysmal atrioventricular block (author's transl)].
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An ajmaline test was conducted in 120 patients with a history of disorders of consciousness : Adams-Stokes syndrome (n = 49), loss of consciousness (n = 42), or lipothymia (n = 29). Four types of response were observed after ajmaline : VH less than 80 ms (n = 63); VH between 80 and 100 ms (n = 19);
[Myocardial infarction in a man with Wolff-Parkinson-White syndrome].
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A case of 52 years old male patient with antero-septal myocardial infarction in the course of type B Wolff-Parkinson-White syndrome has been described. Intravenous administration of 50 mg of ajmaline 2 hours after admission to hospital suppressed the electrocardiographic signs of preexcitation and
[Long term electrophysiological study on patients with acute myocardial infarction and complete heart block. Is cardiac pacing always necessary? (author's transl)].
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Of 335 consecutive patients (pts) admitted to the coronary care unit (CCU) for acute myocardial infarction (AMI), 34 (10%) evidenced complete heart block (HB). The overall inhospital mortality was 14% (47 pts) versus 39% of the HB group (13 pts). No previous conduction disturbances were documented
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