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The effect of high-fructose corn syrup consumption on triglycerides and uric acid.

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Logáil Isteach / Cláraigh

Rates of overweight and obesity have been on a steady rise for decades, and the problems society faces from this and associated metabolic diseases are many. As a result, the need to understand the contributing factors is great. A very compelling case can be made that excess sugar consumption has

Effects on uric acid, body mass index and blood pressure in adolescents of consuming beverages sweetened with high-fructose corn syrup.

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Logáil Isteach / Cláraigh

High-fructose corn syrup and sucrose have equivalent effects on energy-regulating hormones at normal human consumption levels.

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Logáil Isteach / Cláraigh

Intake of high-fructose corn syrup (HFCS) has been suggested to contribute to the increased prevalence of obesity, whereas a number of studies and organizations have reported metabolic equivalence between HFCS and sucrose. We hypothesized that HFCS and sucrose would have similar effects on

Membrane Stabilization and Detoxification of Acetaminophen-Mediated Oxidative Onslaughts in the Kidneys of Wistar Rats by Standardized Fraction of Zea mays L. (Poaceae), Stigma maydis.

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Logáil Isteach / Cláraigh

This study evaluated membrane stabilization and detoxification potential of ethyl acetate fraction of Zea mays L., Stigma maydis in acetaminophen-induced oxidative onslaughts in the kidneys of Wistar rats. Nephrotoxic rats were orally pre- and posttreated with the fraction and vitamin C for 14 days.

Protective effects of aspirin and vitamin C against corn syrup consumption-induced cardiac damage through sirtuin-1 and HIF-1α pathway.

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Logáil Isteach / Cláraigh

The aim of this study was to investigate the protective effects of aspirin (AS) and vitamin C (VC) against cardiac damage induced by chronic corn syrup (CS) consumption via a mechanism involving sirtuin-1 (ST-1), hypoxia-inducible factor-1α (HIF-1α), and the caspase-3 pathway in rats. Forty male

The solute specificity profiles of nucleobase cation symporter 1 (NCS1) from Zea mays and Setaria viridis illustrate functional flexibility.

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Logáil Isteach / Cláraigh

The solute specificity profiles (transport and binding) for the nucleobase cation symporter 1 (NCS1) proteins, from the closely related C4 grasses Zea mays and Setaria viridis, differ from that of Arabidopsis thaliana and Chlamydomonas reinhardtii NCS1. Solute specificity profiles for NCS1 from Z.

High Fructose Corn Syrup Sweetened Soft Drink Consumption Increases Vascular Resistance in the Kidneys at Rest and during Sympathetic Activation.

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Logáil Isteach / Cláraigh

We first tested the hypothesis that consuming a high fructose corn syrup sweetened soft drink (HFCS) augments kidney vasoconstriction to sympathetic stimulation compared to water (Water, Study 1). In a second study, we examined the mechanisms underlying these observations (Study 2). In Study 1,

Hypothesis: could excessive fructose intake and uric acid cause type 2 diabetes?

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Logáil Isteach / Cláraigh

We propose that excessive fructose intake (>50 g/d) may be one of the underlying etiologies of metabolic syndrome and type 2 diabetes. The primary sources of fructose are sugar (sucrose) and high fructose corn syrup. First, fructose intake correlates closely with the rate of diabetes worldwide.

Zea mays L. extracts modify glomerular function and potassium urinary excretion in conscious rats.

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Logáil Isteach / Cláraigh

Diuretic and uricosuric properties have traditionally been attributed to corn silk, stigma/style of Zea mays L. Although the diuretic effect was confirmed, studies of the plant's effects on renal function or solute excretion were lacking. Thus, we studied the effects of corn silk aqueous extract on

Fructose-Rich Beverage Intake and Central Adiposity, Uric Acid, and Pediatric Insulin Resistance.

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Logáil Isteach / Cláraigh

OBJECTIVE To determine the association between sugar-sweetened beverage (SSB) consumption with biomarkers of insulin resistance (IR) and investigate whether/how this relates to obesity and serum uric acid in adolescents. METHODS Adolescents (n = 1454, aged 12-16 years) were assessed in a study

Effects of high-fructose corn syrup and sucrose on the pharmacokinetics of fructose and acute metabolic and hemodynamic responses in healthy subjects.

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Logáil Isteach / Cláraigh

It is unclear whether high-fructose corn syrup (HFCS), which contains a higher amount of fructose and provides an immediate source of free fructose, induces greater systemic concentrations of fructose as compared with sucrose. It is also unclear whether exposure to higher levels of fructose leads to

Fructose containing sugars do not raise blood pressure or uric acid at normal levels of human consumption.

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Logáil Isteach / Cláraigh

The impact of fructose, commonly consumed with sugars by humans, on blood pressure and uric acid has yet to be defined. A total of 267 weight-stable participants drank sugar-sweetened milk every day for 10 weeks as part of their usual, mixed-nutrient diet. Groups 1 and 2 had 9% estimated caloric

Energy and fructose from beverages sweetened with sugar or high-fructose corn syrup pose a health risk for some people.

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Logáil Isteach / Cláraigh

Sugar intake in the United States has increased by >40 fold since the American Revolution. The health concerns that have been raised about the amounts of sugar that are in the current diet, primarily as beverages, are the subject of this review. Just less than 50% of the added sugars (sugar and

A dose-response study of consuming high-fructose corn syrup-sweetened beverages on lipid/lipoprotein risk factors for cardiovascular disease in young adults.

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Logáil Isteach / Cláraigh

BACKGROUND National Health and Nutrition Examination Survey data show an increased risk of cardiovascular disease (CVD) mortality with an increased intake of added sugar. OBJECTIVE We determined the dose-response effects of consuming beverages sweetened with high-fructose corn syrup (HFCS) at zero,

A causal role for uric acid in fructose-induced metabolic syndrome.

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Logáil Isteach / Cláraigh

The worldwide epidemic of metabolic syndrome correlates with an elevation in serum uric acid as well as a marked increase in total fructose intake (in the form of table sugar and high-fructose corn syrup). Fructose raises uric acid, and the latter inhibits nitric oxide bioavailability. Because

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