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6 rezultatima
CADASIL and ALS: a link?
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Prijava Registriraj se
We report the case of a 66-year-old female who presented with dysarthria and dysphonia. Brain MRI abnormalities showed confluent white matter lesions and subcortical lacunar infarcts, suggesting cerebral autosomal dominant arteriopathy with subcortical infarcts and leucoencephalopathy (CADASIL),
Neuromuscular implications in CADASIL.
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Prijava Registriraj se
OBJECTIVE
Recent studies indicate that Notch3 gene mutations not only manifest as cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) but also in the peripheral nerves and skeletal muscles.
METHODS
A MEDLINE search with appropriate terms was carried
CADASIL accelerated by acute hypotension: Arterial and venous contribution to leukoaraiosis.
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Prijava Registriraj se
OBJECTIVE
To underline the importance of blood pressure regulation in cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) and to describe changes that occur in the veins in this condition, specifically venous collagenosis associated with
Non-CAA angiopathies and their possible interactions with cerebral amyloid angiopathy.
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Prijava Registriraj se
Cerebral amyloid angiopathy (CAA) is one of the two most common cerebral arteriopathies seen in the brains of elderly patients. The other is arteriosclerosis (AS), historically considered a consequence of chronic hypertension and also described as lipohyalinosis (LH), a clinicopathologic association
Cognitive impairment and cellular/vascular changes in the cerebral white matter.
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Prijava Registriraj se
A possible relation between cerebral white-matter injury and dementia was intuitively attributed by Alzheimer to changes affecting the small penetrating vessels that supply the cerebral white matter. Several observations support the view that white-matter changes detectable by neuroimaging may
Neuropathology of cerebrovascular diseases.
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Prijava Registriraj se
The chapter describes the epidemiology of cerebrovascular diseases, anatomy of the cerebral blood vessels, pathophysiology of ischemia, hypoxia, hypoxemia, anemic hypoxia, histotoxic hypoxia, carbon monoxide damage, hyperoxid brain damage and decompression sickness, and selective cell and regional
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