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Stranica 1 iz 151 rezultatima
Suppression of catatonia-like signs by lorazepam in nonconvulsive status epilepticus without seizure termination.
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Nonconvulsive status epilepticus (NCSE) cannot be reliably distinguished from catatonia by signs or symptoms. The authors report on a 68-year-old man with endogenous major depression who displayed catatonia-like psychopathology, which temporarily disappeared with lorazepam. An EEG then revealed
Catatonia in encephalitis and nonconvulsive seizures: a case report and review of the literature.
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A 20-year-old woman was admitted for psychosis. On further investigation, she was found to be have viral encephalitis and generalized nonconvulsive seizures. After the seizures were controlled, she remained in a prolonged catatonic state. Repeated intravenous benzodiazepine administration, improved
Persistent catatonia following epileptic seizures: a case report and systematic literature search.
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BACKGROUND
Catatonia is frequently associated with mood and psychotic disorders as well as with general medical conditions, especially with seizures. In the case of the latter, catatonia mostly resolves when the seizures respond to the anticonvulsive treatment. We report, to our knowledge, the first
Resolution of catatonia by successful seizure induction via electroconvulsive therapy with electrodes applied bilaterally to the parietotemporal region.
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Electroconvulsive therapy (ECT) has been shown to be effective in cases of medically intractable mood disorder and schizophrenia. However, some patients receiving ECT have only a short electroencephalographic seizure or no seizure at the maximum stimulus intensity and thus fail to obtain a
Impact of High Dose Lorazepam on Seizure Threshold in Catatonia: Experience from a Case Study.
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On catatonia, seizures, and bradycardia.
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Resting-state hyperperfusion in the whole brain: A case of malignant catatonia that improved with electric convulsion therapy.
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THE EFFECT OF CARDIAZOL CONVULSIONS ON THE SO-CALLED "BULBOCAPNINE CATATONIA" IN THE MONKEY.
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Resolution of self-injury with phenytoin in a man with autism and intellectual disability: the role of frontal lobe seizures and catatonia.
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Successful treatment of anti-N-methyl-D-aspartate receptor encephalitis presenting with catatonia.
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The case of a 12-year-old girl with the typical clinical symptoms of the recently described anti-N-methyl-D-aspartate receptor (NMDAR) encephalitis is reported. Within 6 weeks the full clinical spectrum of this condition presented with seizures, agitation, stupor, autonomic instability, dysphagia
The Probability That Catatonia in the Hospital has a Medical Cause and the Relative Proportions of Its Causes: A Systematic Review.
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OBJECTIVE
The objective of this review is to determine the probability that catatonia in the hospital has a secondary cause ("medical catatonia") and to calculate the relative proportions of these causes stratified by hospital setting.
METHODS
PRISMA systematic review of PubMed.
RESULTS
Eleven
Catatonia following surgery for temporal lobe epilepsy successfully treated with electroconvulsive therapy.
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Catatonia is a psychomotor and behavioral syndrome associated with mood disorders, psychotic disorders, neurological disease, and general medical conditions. Catatonia has also been described as a manifestation of partial onset seizures. We describe a case of catatonia following successful epilepsy
Ictal catatonia as a manifestation of nonconvulsive status epilepticus.
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Three patients with EEG documented ictal catatonia, a nonconvulsive status epilepticus, who responded dramatically to intravenous phenytoin are described. The EEG showed continuous bilateral pseudoperiodic sharp waves and spike discharges in one patient, spike and wave complexes were seen
[Anti-NMDA encephalitis in psychiatry; malignant catatonia, atypical psychosis and ECT].
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The symptoms of malignant (lethal) catatonia has been reported similar to initial symptoms of anti-NMDAR encephalitis. Subsequently, this autoimmune limbic encephalitis has been noticed in many psychiatrists. We have experienced several cases with malignant catatonia having anti-NMDAR antibody
Intermittent catatonia and complex automatisms caused by frontal lobe epilepsy in dementia.
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An 82-year-old man was admitted to the emergency department following bizarre behaviour. Police had noticed him driving erratically through his village. He did not stop when instructed, drove slowly home and appeared 'vacant' on questioning. While in hospital, he had approximately 15 episodes of
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