embelia sessiliflora/phosphatase

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Histochemical localization of alkaline phosphatase in the uterus of rats: response to a few indigenous plant extracts.

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Localization of alkaline phosphatase in the uterine luminal and glandular epithelium of rats under the influence of 50% ethanolic and benzene extracts of three indigenous plants viz. Embelia ribes Burm. (dried berries), Artobotrys odoratissimus Linn. (fresh green leaves) and Hibiscus rosasinensis

Phosphatases activity in the uterus of intact and ovariectomized rats: response to Embelia ribes extracts.

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Embelin can protect mice from thioacetamide-induced acute liver injury.

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Embelin is an active component isolated from Embelia ribes Burm. In this study, we explored the protective effects of embelin on acute liver injury.An animal model of acute liver injury was established via administration of a single injection of

Chemopreventive and hepatoprotective effects of embelin on N-nitrosodiethylamine and carbon tetrachloride induced preneoplasia and toxicity in rat liver.

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Embelin, an active constituent isolated from the fruits of Embelia tsjeriam cottam was investigated for its chemopreventive and hepatoprotective effects against N-nitrosodiethylamine (NDEA) induced liver preneoplasia or carbon tetrachloride (CCl4) induced liver damage. Rats received NDEA, 1 ppm/g

Embelia ribes extract reduces high fat diet and low dose streptozotocin-induced diabetic nephrotoxicity in rats.

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Nephropathy associated with type 2 diabetes is the single most common cause of end-stage renal disease. The aim of the present study was to evaluate the preventive effect of ethanolic extract of Embelia ribes fruit (EER) against high fat diet (HFD) and low dose streptozotocin (STZ)-induced diabetic

Effects of embelin on lipid peroxidation and free radical scavenging activity against liver damage in rats.

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The study aimed to evaluate the hepatic antioxidant capacity of embelin (from Embelia ribes) using different antioxidant tests, free radical scavenging activity and lipid peroxidation in albino rats. Carbon tetrachloride (CCl₄) treatment to rats has been more susceptible to peroxidative damage

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