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epilepsia partialis continua/atrophy
Veza se sprema u međuspremnik
Stranica 1 iz 72 rezultatima
Progressive cerebral degeneration of childhood with liver disease (Alpers Huttenlocher disease) with cytochrome oxidase deficiency presenting with epilepsia partialis continua as the first clinical manifestation.
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A previously healthy and normally developed 17-year-old young female presented with a sudden onset of focal motor seizure status that proved to be refractory to anticonvulsive treatment. Severe encephalopathy with visual impairment leading to blindness, mental deterioration, and predominantly left
Benzodiazepine receptor imaging in an adult with Rasmussen's encephalitis and epilepsia partialis continua.
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We describe an adult with Rasmussen's encephalitis associated with widespread decreased accumulation of tracer on 123-I iomazenil (IMZ) SPECT. The patient had a form of epilepsia partialis continua and neurological deterioration associated with hemiatrophy. Cranial MRI showed cerebral atrophy in the
Epilepsia partialis continua of childhood involving bilateral brain hemispheres.
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OBJECTIVE
Epilepsia partialis continua (Kojewnikow's syndrome) can be classified into 2 groups. The 1st group had stable neurological deficit, and the 2nd group had slowly progressive neurological deficit. The latter usually manifests not tetraplegia, but hemiplegia. We describe 3 patients with
Epilepsia partialis continua following pertussis infection.
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Clinical, electrophysiological and CT findings of a patient with epilepsia partialis continua following pertussis infection are presented. The EEG showed lateralized abnormal slow waves and CT revealed global atrophy around the Sylvian fissure. The findings favor the primary role of the cerebral
Intractable focal epilepsy contralateral to the side of facial atrophy in Parry-Romberg syndrome.
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We report an unusual case of Parry-Romberg syndrome in which medically refractory focal epilepsy with ongoing epilepsia partialis continua (EPC) arose from the hemisphere contralateral to the side of facial atrophy. Unilateral cerebral involvement was confirmed by multi-modal brain imaging, as well
[Nosological aspects of epilepsia partialis continua in children].
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Among 26 patients suffering from Epilepsia Partialis Continua, 2 major groups were observed. The first, resulting from a fixed lesion of the rolandic area, showed electro-clinical correlation of seizures; the latter disappeared during sleep; clinical and radiological follow-up failed to disclose any
Epilepsia partialis continua due to cortical dysplasia.
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We describe a child with progressive neurologic deterioration and epilepsia partialis continua of the right extremities. Magnetic resonance imaging suggested cortical dysplasia of the left frontal region including the central sensorimotor cortex. Subdural grid recordings showed widespread ictal
Kufs' disease presenting as late-onset epilepsia partialis continua.
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A 64-year-old woman, who had no personal or family history of neurologic diseases, had an 18-month history of epilepsia partialis continua (EPC) associated with a moderate intellectual deterioration and subtle extrapyramidal rigidity. There was no photosensitive response. A thorough laboratory
Epilepsia partialis continua: follow-up with 99mTc-HMPAO-SPECT.
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A three-year-old girl with epilepsia partialis continua involving the left side of the face and the left arm and leg was followed-up by 99mTc-HMPAO-SPECT in addition to EEG and CT. Increased regional cerebral blood flow was found in the right Rolandic area in the early stage of the disease, when the
Epilepsia partialis continua as an isolated manifestation of motor cortical dysplasia.
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Cortical dysplasia has been increasingly recognized as a cause of epilepsy. We describe herein a 31-year-old female patient with epilepsia partialis continua (EPC) in the right extremities, which had lasted for 15 years without generalized seizures and other neurological deteriorations. MRI showed a
Familial alternating epilepsia partialis continua with chronic encephalitis: another variant of Rasmussen syndrome?
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Two brothers had infantile epilepsia partialis continua alternately involving both sides of the body. The children rapidly developed severe psychomotor regression and cerebral atrophy. A brain biopsy specimen showed evidence of chronic inflammatory changes. Extensive investigation did not provide
Epilepsia partialis continua in children with fulminant subacute sclerosing panencephalitis.
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Various inflammatory diseases of central nervous system, including subacute sclerosing panencephalitis, could cause epilepsia partialis continua. Two boys with epilepsia partialis continua with onset in terminal phase of atypical subacute sclerosing panencephalitis have been reported. Children were
Epilepsia partialis continua and generalized nonconvulsive status epilepticus during the course of argininemia: a report on two cases.
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Argininemia is a rare inherited disorder of the urea cycle because of a deficiency of the enzyme arginase I causing an increase of arginine and guanidino compounds in the blood, urine, and cerebrospinal fluid. The clinical picture is characterized by a mild cognitive dysfunction, progressive
Anomalies of the cerebral cortex in a case of epilepsia partialis continua.
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Morphological and biochemical findings are described from the brain biopsy of an 11-year-old girl with intractable type II epilepsia partialis continua. Computerized tomography scan showed severe progressive central and cortical atrophy, mainly of the right hemisphere. Brain biopsy revealed
[A variant of the epilepsia partialis continua syndrome in a child with hypoplasia of the internal carotid artery].
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The authors describe a case of a 13-year-old adolescent. Being seven years old, he developed the epileptic syndrome associated with persistent focal and secondary generalized fits refractory to the treatment. Later the clinical picture became very suggestive of the syndrome Epilepsia partialis
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