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glucose 6 phosphate dehydrogenase/groznica
Veza se sprema u međuspremnik
Stranica 1 iz 120 rezultatima
Haemolysis with Mediterranean spotted fever and glucose-6-phosphate dehydrogenase deficiency.
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Mediterranean spotted fever with haemolysis is reported in a glucose-6-phosphate dehydrogenase deficient Algerian man. The clinical course was unusually severe for a 27-year-old patient. The authors suspect G6PD deficiency to be a cause of enhanced severity in Mediterranean spotted fever as well as
Effect of red blood cell glucose-6-phosphate dehydrogenase deficiency on patients with dengue hemorrhagic fever.
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Eighty nine males aged 1-13 years diagnosed with dengue haemorrhagic fever (DHF) and admitted to the Department of Pediatrics Siriraj Hospital from March 1998 to April 2000 were included in this study. 17 cases (19.1%) had red blood cell glucose-6-phosphate dehydrogenase (G-6-PD) deficiency and 72
Isoflurane Induced Malignant Hyperthermia in a Patient with Glucose 6-Phosphate Dehydrogenase Deficiency and Growth Hormone Abuse
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Malignant hyperthermia is a pharmacogenetic disorder in the regulation of calcium in skeletal muscles which is related to an uninhibited muscle hypermetabolic reaction to potent inhalation agents, the depolarizing muscle relaxant succinylcholine, and to stressors such as vigorous exercise and heat.
Canine malignant hyperthermia susceptibility: erythrocytic defects--osmotic fragility, glucose-6-phosphate dehydrogenase deficiency and abnormal Ca2+ homeostasis.
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Two dogs were diagnosed as malignant hyperthermia susceptible based on increased susceptibility (P less than 0.001) of biopsied muscle to caffeine-induced contracture. Erythrocytes from malignant hyperthermia and normal dogs were then examined for an antioxidant system deficiency. Values for serum
Typhoid fever and haemolytic anaemia in a black patient with glucose-6-phosphate dehydrogenase deficiency. A case report.
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A young Zulu man was admitted for investigation of anaemia, jaundice and fever. He had a haemolytic anaemia, glucose-6-phosphate dehydrogenase (G6PD) deficiency and typhoid fever. Reports on haemolysis as a complication of typhoid fever in patients with G6PD deficiency are exceedingly rare in
Depression of erythrocyte glucose-6-phosphate dehydrogenase (G6PD) activity in enteric fever.
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Ninety adult Indian typhoid and paratyphoid fever (enteric fever, EF) patients and 91 controls were tested for glucose-6-phosphate dehydrogenase (G6PD) deficiency using the fluorescent spot test (FST) and the quantitative methaemoglobin reduction test ( QMRT ). There was a threefold higher incidence
Fulminant Rocky Mountain spotted fever. Its pathologic characteristics associated with glucose-6-phosphate dehydrogenase deficiency.
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Three patients with documented fulminant Rocky Mountain spotted fever (RMSF) (death on or before day 5 of illness) had severe multisystemic injury as shown by clinical signs and laboratory data, but on microscopic examination showed minimal evidence of the typical mononuclear leukocytic response to
Transient, acquired glucose-6-phosphate dehydrogenase deficiency in Thai children with typhoid fever.
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Seventy nine children with typhoid fever were studied, 45 were males and 34 were females. There were 9 out of 45 males patients (20%) who had definite G-6-PD deficiency of whom 3 had acute intravascular hemolysis. The rest of the patients had transient low enzyme activity during the first few weeks
[Hepatic and myocardial glucokinase, hexokinase and glucose-6-phosphate dehydrogenase activity in rabbits with pyrogenal fever].
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The activity of glucokinase, hexokinase and glucose-6- phosphoric dehydrogenase of the liver and myocardium of rabbits was tested at different stages of pyrogenal fever with the aid of spectrophotometry. A marked decrease in the activity of the enzymes under study was observed in fever. After the
Serum transcobalamin II level in glucose-6-phosphate dehydrogenase deficient subjects with typhoid fever.
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Transcobalamin II (TCII) is the vitamin B12 binding protein which is responsible for delivery of this vitamin to the tissues. High values for serum TCII have been reported in many clinical conditions. This paper describes the elevated serum TCII levels in three G-6-PD deficient patients with typhoid
Co-trimoxazole in the treatment of typhoid fever in children with glucose-6-phosphate dehydrogenase deficiency.
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The efficacy and possible adverse reactions of co-trimoxazole in the treatment of typhoid fever with G-6-PD deficiency were investigated in 68 typhoid children aged 2 to 14 years old. Salmonella typhi was isolated from 45 patients but all had a significant rise of Widal agglutinin titres during the
Frequency of glucose-6-phosphate dehydrogenase (G-6-PD) deficiency in Tanzanian males with typhoid fever.
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The frequency of G-6-PD deficiency in the Dar es Salaam male population is given as 18.7%. In this preliminary study, is reported a frequency of 45% in males with typhoid fever--a figure more than twice the prevalence in the general Dar es Salaam male population. Possible reasons for positive
[A case of typhoid fever in a visitor from India with glucose-6-phosphate dehydrogenase deficiency. Remarkable conjugated hyperbilirubinemia probably induced by an analgesic drug (author's transl)].
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Malignant hyperthermia and glucose-6-phosphate dehydrogenase deficiency.
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The survival of glucose-6-phosphate dehydrogenase--deficient erythrocytes in patients with typhoid fever on chloramphenicol therapy.
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